16083698

Source:http://linkedlifedata.com/resource/pubmed/id/16083698

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0039082, umls-concept:C0206132, umls-concept:C0596244, umls-concept:C1333990
pubmed:issue	2
pubmed:dateCreated	2005-8-8
pubmed:abstractText	Mutations in the mismatch repair genes cause hereditary nonpolyposis colorectal cancer (HNPCC) syndrome and convey high lifetime cancer risks for colorectal (CRC) and endometrial cancer. Currently, cancer risks for individuals with HNPCC are based on data from clinically ascertained families. The purpose of this study was to re-examine the penetrance in HNPCC using a comprehensive dataset from a geographically defined region.
pubmed:grant	http://linkedlifedata.com/resource/pubmed/grant/CA67941, http://linkedlifedata.com/resource/pubmed/grant/P30 CA16058
pubmed:commentsCorrections	http://linkedlifedata.com/resource/pubmed/commentcorrection/16083698-16083727
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/0374630
pubmed:citationSubset	AIM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Adaptor Proteins, Signal Transducing, http://linkedlifedata.com/resource/pubmed/chemical/Carrier Proteins, http://linkedlifedata.com/resource/pubmed/chemical/MLH1 protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Neoplasm Proteins, http://linkedlifedata.com/resource/pubmed/chemical/Nuclear Proteins
pubmed:status	MEDLINE
pubmed:month	Aug
pubmed:issn	0016-5085
pubmed:author	pubmed-author:AaltonenLauri ALA, pubmed-author:HampelHeatherH, pubmed-author:MecklinJukka-PekkaJP, pubmed-author:PukkalaEeroE, pubmed-author:SankilaRistoR, pubmed-author:StephensJulie AJA, pubmed-author:de la ChapelleAlbertA
pubmed:issnType	Print
pubmed:volume	129
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	415-21
pubmed:dateRevised	2009-11-19
pubmed:meshHeading	pubmed-meshheading:16083698-Adaptor Proteins, Signal Transducing, pubmed-meshheading:16083698-Age Distribution, pubmed-meshheading:16083698-Age of Onset, pubmed-meshheading:16083698-Aged, pubmed-meshheading:16083698-Aged, 80 and over, pubmed-meshheading:16083698-Carrier Proteins, pubmed-meshheading:16083698-Cohort Studies, pubmed-meshheading:16083698-Colorectal Neoplasms, pubmed-meshheading:16083698-Colorectal Neoplasms, Hereditary Nonpolyposis, pubmed-meshheading:16083698-Confidence Intervals, pubmed-meshheading:16083698-DNA Mutational Analysis, pubmed-meshheading:16083698-Female, pubmed-meshheading:16083698-Gene Expression Regulation, Neoplastic, pubmed-meshheading:16083698-Genetic Testing, pubmed-meshheading:16083698-Humans, pubmed-meshheading:16083698-Male, pubmed-meshheading:16083698-Middle Aged, pubmed-meshheading:16083698-Neoplasm Proteins, pubmed-meshheading:16083698-Neoplasm Staging, pubmed-meshheading:16083698-Nuclear Proteins, pubmed-meshheading:16083698-Precancerous Conditions, pubmed-meshheading:16083698-Probability, pubmed-meshheading:16083698-Prognosis, pubmed-meshheading:16083698-Risk Assessment, pubmed-meshheading:16083698-Sex Distribution, pubmed-meshheading:16083698-Survival Analysis
pubmed:year	2005
pubmed:articleTitle	Cancer risk in hereditary nonpolyposis colorectal cancer syndrome: later age of onset.
pubmed:affiliation	Human Cancer Genetics Program, The Ohio State University Comprehensive Cancer Center, The Ohio State University, Columbus, 43210, USA.
pubmed:publicationType	Journal Article, Comparative Study, Comment, Research Support, U.S. Gov't, P.H.S., Research Support, N.I.H., Extramural