Source:http://linkedlifedata.com/resource/pubmed/id/16033093
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
3A
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pubmed:dateCreated |
2005-7-21
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pubmed:abstractText |
p53 inactivation due to oncogenic viral proteins or mutations is an important molecular mechanism in carcinogenesis, which has also been demonstrated for vulvar carcinoma. To evaluate p53 changes in vulvar carcinogenesis, we analyzed p53 expression in vulvar carcinoma, vulvar intraepithelial neoplasia (VIN), lichen sclerosus (LS) and squamous cell hyperplasia (SH).
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:issn |
0250-7005
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
25
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1739-45
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pubmed:dateRevised |
2005-11-17
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pubmed:meshHeading |
pubmed-meshheading:16033093-Carcinoma in Situ,
pubmed-meshheading:16033093-Female,
pubmed-meshheading:16033093-Humans,
pubmed-meshheading:16033093-Hyperplasia,
pubmed-meshheading:16033093-Immunohistochemistry,
pubmed-meshheading:16033093-Lichen Sclerosus et Atrophicus,
pubmed-meshheading:16033093-Middle Aged,
pubmed-meshheading:16033093-Tumor Suppressor Protein p53,
pubmed-meshheading:16033093-Vulvar Neoplasms
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pubmed:articleTitle |
P53 expression in vulvar carcinoma, vulvar intraepithelial neoplasia, squamous cell hyperplasia and lichen sclerosus.
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pubmed:affiliation |
Department of Gynecology and Obstetrics, Kreisklinik Altötting, 84503 Altötting, Germany.
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pubmed:publicationType |
Journal Article
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