Source:http://linkedlifedata.com/resource/pubmed/id/16004800
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
7
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pubmed:dateCreated |
2005-7-11
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pubmed:abstractText |
Low level expression of some co-stimulatory molecules is an important mechanism for tumor to evade the attack of immunity system; up-regulating these molecules can enhance antitumor immune response. Mouse melanoma B16-F1 cell line is low immunogenic. This study was designed to investigate the inhibitory effect of co-stimulatory molecule 4-1BBL combined with hot shock protein 70 (Hsp70)-tumor antigen peptides on pulmonary metastasis of melanoma in mice, and to analyze the possible mechanism.
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pubmed:language |
chi
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Jul
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pubmed:issn |
1000-467X
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
24
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
781-6
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pubmed:dateRevised |
2008-11-21
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pubmed:meshHeading |
pubmed-meshheading:16004800-4-1BB Ligand,
pubmed-meshheading:16004800-Animals,
pubmed-meshheading:16004800-CD8-Positive T-Lymphocytes,
pubmed-meshheading:16004800-Cell Line, Tumor,
pubmed-meshheading:16004800-HSP70 Heat-Shock Proteins,
pubmed-meshheading:16004800-Interferon-gamma,
pubmed-meshheading:16004800-Interleukin-2,
pubmed-meshheading:16004800-Liver,
pubmed-meshheading:16004800-Lung Neoplasms,
pubmed-meshheading:16004800-Male,
pubmed-meshheading:16004800-Melanoma, Experimental,
pubmed-meshheading:16004800-Mice,
pubmed-meshheading:16004800-Mice, Inbred BALB C,
pubmed-meshheading:16004800-Plasmids,
pubmed-meshheading:16004800-Transfection
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pubmed:year |
2005
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pubmed:articleTitle |
[Inhibitory effect of 4-1BBL combined with Hsp70-tumor antigen peptides on pulmonary metastasis of melanoma in mice].
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pubmed:affiliation |
Department of Biochemistry and Molecular Biology, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei, 430030, PR China.
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pubmed:publicationType |
Journal Article,
English Abstract,
Research Support, Non-U.S. Gov't
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