pubmed-article:15986373 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:15986373 | lifeskim:mentions | umls-concept:C0087111 | lld:lifeskim |
pubmed-article:15986373 | lifeskim:mentions | umls-concept:C0754728 | lld:lifeskim |
pubmed-article:15986373 | lifeskim:mentions | umls-concept:C0949691 | lld:lifeskim |
pubmed-article:15986373 | lifeskim:mentions | umls-concept:C1411976 | lld:lifeskim |
pubmed-article:15986373 | lifeskim:mentions | umls-concept:C1456820 | lld:lifeskim |
pubmed-article:15986373 | lifeskim:mentions | umls-concept:C0205276 | lld:lifeskim |
pubmed-article:15986373 | lifeskim:mentions | umls-concept:C0205217 | lld:lifeskim |
pubmed-article:15986373 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
pubmed-article:15986373 | lifeskim:mentions | umls-concept:C0332206 | lld:lifeskim |
pubmed-article:15986373 | lifeskim:mentions | umls-concept:C0205373 | lld:lifeskim |
pubmed-article:15986373 | lifeskim:mentions | umls-concept:C2911684 | lld:lifeskim |
pubmed-article:15986373 | lifeskim:mentions | umls-concept:C0185117 | lld:lifeskim |
pubmed-article:15986373 | pubmed:issue | 7 | lld:pubmed |
pubmed-article:15986373 | pubmed:dateCreated | 2005-7-4 | lld:pubmed |
pubmed-article:15986373 | pubmed:abstractText | Abnormal host defense against pathogens has been implicated in the pathogenesis of spondylarthropathy (SpA), a disease characterized by abundant synovial infiltration with innate immune cells. Given the role of Toll-like receptors (TLRs) in activation of innate inflammation and the occurrence of TLR-dependent infections after tumor necrosis factor alpha (TNFalpha) blockade treatment, the present study was undertaken to analyze TLRs and their modulation by TNFalpha blockade in SpA. | lld:pubmed |
pubmed-article:15986373 | pubmed:language | eng | lld:pubmed |
pubmed-article:15986373 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15986373 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:15986373 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15986373 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15986373 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15986373 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15986373 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15986373 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15986373 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15986373 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15986373 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15986373 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15986373 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15986373 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15986373 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15986373 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15986373 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15986373 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:15986373 | pubmed:month | Jul | lld:pubmed |
pubmed-article:15986373 | pubmed:issn | 0004-3591 | lld:pubmed |
pubmed-article:15986373 | pubmed:author | pubmed-author:BaetenDominiq... | lld:pubmed |
pubmed-article:15986373 | pubmed:author | pubmed-author:KruithofElliE | lld:pubmed |
pubmed-article:15986373 | pubmed:author | pubmed-author:VeysEric MEM | lld:pubmed |
pubmed-article:15986373 | pubmed:author | pubmed-author:De... | lld:pubmed |
pubmed-article:15986373 | pubmed:author | pubmed-author:De RyckeLeenL | lld:pubmed |
pubmed-article:15986373 | pubmed:author | pubmed-author:VandoorenBern... | lld:pubmed |
pubmed-article:15986373 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:15986373 | pubmed:volume | 52 | lld:pubmed |
pubmed-article:15986373 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:15986373 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:15986373 | pubmed:pagination | 2146-58 | lld:pubmed |
pubmed-article:15986373 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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pubmed-article:15986373 | pubmed:year | 2005 | lld:pubmed |
pubmed-article:15986373 | pubmed:articleTitle | Tumor necrosis factor alpha blockade treatment down-modulates the increased systemic and local expression of Toll-like receptor 2 and Toll-like receptor 4 in spondylarthropathy. | lld:pubmed |
pubmed-article:15986373 | pubmed:affiliation | Ghent University Hospital, Ghent, Belgium. | lld:pubmed |
pubmed-article:15986373 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:15986373 | pubmed:publicationType | Clinical Trial | lld:pubmed |
pubmed-article:15986373 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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