Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
2005-6-21
pubmed:abstractText
The pathophysiology of acute pancreatitis is strongly associated with autoactivation of trypsin. The biologic activity of trypsin on cells is attributed to the activation of protease-activated receptor-2 (PAR-2). We hypothesize that trypsin may activate acinar cells or inflammatory cells through PAR-2 signals in acute pancreatitis.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jul
pubmed:issn
1536-4828
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
31
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
54-62
pubmed:meshHeading
pubmed:year
2005
pubmed:articleTitle
Proinflammatory role of trypsin and protease-activated receptor-2 in a rat model of acute pancreatitis.
pubmed:affiliation
Department of Gastroenterological Surgery, Postgraduate School of Medicine, Kumamoto University, Kumamoto-city, Kumamoto, Japan.
pubmed:publicationType
Journal Article