| pubmed-article:1596084 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:1596084 | lifeskim:mentions | umls-concept:C0036572 | lld:lifeskim |
| pubmed-article:1596084 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:1596084 | lifeskim:mentions | umls-concept:C0021270 | lld:lifeskim |
| pubmed-article:1596084 | lifeskim:mentions | umls-concept:C0002708 | lld:lifeskim |
| pubmed-article:1596084 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:1596084 | pubmed:dateCreated | 1992-7-2 | lld:pubmed |
| pubmed-article:1596084 | pubmed:abstractText | The neuroanatomical substrate of seizures induced by picomolar amounts of corticotropin-releasing hormone in infant rats was investigated. Electrographic and behavioral phenomena were monitored in 42 rat pups aged 5 to 22 days. Rat pups carried bipolar electrodes implanted in subcortical limbic structures, as well as cortical electrodes and intracerebroventricular cannulae. The administration of corticotropin-releasing hormone produced age-specific seizures within minutes, which correlated with rhythmic amygdala discharges. Paroxysmal hippocampal and cortical discharges developed subsequently in some rats. Corticotropin-releasing hormone-induced electrographic and behavioral seizures originate in the amygdala. | lld:pubmed |
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| pubmed-article:1596084 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:1596084 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:1596084 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1596084 | pubmed:month | May | lld:pubmed |
| pubmed-article:1596084 | pubmed:issn | 0364-5134 | lld:pubmed |
| pubmed-article:1596084 | pubmed:author | pubmed-author:SchultzLL | lld:pubmed |
| pubmed-article:1596084 | pubmed:author | pubmed-author:SneadO COC3rd | lld:pubmed |
| pubmed-article:1596084 | pubmed:author | pubmed-author:HirschEE | lld:pubmed |
| pubmed-article:1596084 | pubmed:author | pubmed-author:BaramT ZTZ | lld:pubmed |
| pubmed-article:1596084 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1596084 | pubmed:volume | 31 | lld:pubmed |
| pubmed-article:1596084 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1596084 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1596084 | pubmed:pagination | 488-94 | lld:pubmed |
| pubmed-article:1596084 | pubmed:dateRevised | 2011-9-13 | lld:pubmed |
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| pubmed-article:1596084 | pubmed:year | 1992 | lld:pubmed |
| pubmed-article:1596084 | pubmed:articleTitle | Corticotropin-releasing hormone-induced seizures in infant rats originate in the amygdala. | lld:pubmed |
| pubmed-article:1596084 | pubmed:affiliation | Department of Neurology, University of Southern California, Los Angeles. | lld:pubmed |
| pubmed-article:1596084 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1596084 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:1596084 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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