rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
5
|
pubmed:dateCreated |
2005-6-10
|
pubmed:abstractText |
Rapid administration of suboptimal antigen induces transient unresponsiveness in patients with IgE antibodies to beneficial medications, but the molecular mechanisms of desensitization are poorly understood. Mast cells (MCs) have been implicated as the target cells.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
IM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
May
|
pubmed:issn |
1081-1206
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
94
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
575-80
|
pubmed:dateRevised |
2006-11-15
|
pubmed:meshHeading |
pubmed-meshheading:15945561-Animals,
pubmed-meshheading:15945561-Antibodies, Monoclonal,
pubmed-meshheading:15945561-Antigens,
pubmed-meshheading:15945561-Bone Marrow,
pubmed-meshheading:15945561-Desensitization, Immunologic,
pubmed-meshheading:15945561-Dinitrobenzenes,
pubmed-meshheading:15945561-Dose-Response Relationship, Immunologic,
pubmed-meshheading:15945561-Immunization,
pubmed-meshheading:15945561-Mast Cells,
pubmed-meshheading:15945561-Mice,
pubmed-meshheading:15945561-Mice, Inbred BALB C,
pubmed-meshheading:15945561-Mice, Inbred C57BL,
pubmed-meshheading:15945561-Picrates,
pubmed-meshheading:15945561-STAT6 Transcription Factor,
pubmed-meshheading:15945561-Time Factors,
pubmed-meshheading:15945561-Trans-Activators
|
pubmed:year |
2005
|
pubmed:articleTitle |
Antigen-IgE desensitization in signal transducer and activator of transcription 6-deficient mast cells by suboptimal doses of antigen.
|
pubmed:affiliation |
Department of Medicine, Harvard Medical School and Brigham and Women's Hospital, Boston, Massachusetts 02115, USA.
|
pubmed:publicationType |
Journal Article,
Comparative Study,
Research Support, Non-U.S. Gov't
|