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pubmed-article:15939815pubmed:abstractTextAlthough disruption of guanylyl cyclase (GC) A, a natriuretic peptide receptor, induces cardiac hypertrophy and fibrosis, the molecular mechanism underlying these effects are not well understood. In this study, we examined the role of calcineurin, a calcium-dependent phosphatase, in cardiac remodeling in GCA-knockout (GCA-KO) mice.lld:pubmed
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pubmed-article:15939815pubmed:articleTitleCalcineurin-nuclear factor of activated T cells pathway-dependent cardiac remodeling in mice deficient in guanylyl cyclase A, a receptor for atrial and brain natriuretic peptides.lld:pubmed
pubmed-article:15939815pubmed:affiliationDepartment of Biochemistry, Research Institute, National Cardiovascular Center, Suita, Osaka, Japan.lld:pubmed
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