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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
5
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pubmed:dateCreated |
2005-6-1
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pubmed:abstractText |
Hypertrophic cardiomyopathy (HCM) is a genetic-based disease. Several gene mutations leading to HCM development have been described.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
May
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pubmed:issn |
0022-9032
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
62
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
440-9; 449-50
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pubmed:meshHeading |
pubmed-meshheading:15928721-Adolescent,
pubmed-meshheading:15928721-Adult,
pubmed-meshheading:15928721-Aged,
pubmed-meshheading:15928721-Cardiomyopathy, Hypertrophic, Familial,
pubmed-meshheading:15928721-Female,
pubmed-meshheading:15928721-Gene Deletion,
pubmed-meshheading:15928721-Genotype,
pubmed-meshheading:15928721-Humans,
pubmed-meshheading:15928721-Male,
pubmed-meshheading:15928721-Middle Aged,
pubmed-meshheading:15928721-Pedigree,
pubmed-meshheading:15928721-Peptidyl-Dipeptidase A,
pubmed-meshheading:15928721-Phenotype,
pubmed-meshheading:15928721-Polymorphism, Genetic,
pubmed-meshheading:15928721-Receptor, Angiotensin, Type 1,
pubmed-meshheading:15928721-Receptors, Angiotensin
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pubmed:year |
2005
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pubmed:articleTitle |
Familial hypertrophic cardiomyopathy. Insertion-deletion polymorphism of angiotensin-converting enzyme and angiotensin II receptor.
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pubmed:affiliation |
2nd chair and Department of Cardiology, Zabrze.
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pubmed:publicationType |
Journal Article
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