| pubmed-article:15927991 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:15927991 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:15927991 | lifeskim:mentions | umls-concept:C0020542 | lld:lifeskim |
| pubmed-article:15927991 | lifeskim:mentions | umls-concept:C0170657 | lld:lifeskim |
| pubmed-article:15927991 | lifeskim:mentions | umls-concept:C0021467 | lld:lifeskim |
| pubmed-article:15927991 | lifeskim:mentions | umls-concept:C1555029 | lld:lifeskim |
| pubmed-article:15927991 | lifeskim:mentions | umls-concept:C0021469 | lld:lifeskim |
| pubmed-article:15927991 | lifeskim:mentions | umls-concept:C1292733 | lld:lifeskim |
| pubmed-article:15927991 | pubmed:issue | 21 | lld:pubmed |
| pubmed-article:15927991 | pubmed:dateCreated | 2005-6-1 | lld:pubmed |
| pubmed-article:15927991 | pubmed:abstractText | Progression of pulmonary hypertension (PH) is associated with increased lung expression of the serotonin transporter (5-HTT), which leads to hyperplasia of the pulmonary artery smooth muscle cells (PA-SMCs). Given the postulated causal relation between 5-HTT overexpression and PH, we herein investigated whether the highly selective 5-HTT inhibitor fluoxetine prevented and/or reversed PH induced by monocrotaline (MCT) in rats. Selective 5-HT(1B/1D), 5-HT(2A), and 5-HT(2B) receptor antagonists were used for comparative testing. | lld:pubmed |
| pubmed-article:15927991 | pubmed:language | eng | lld:pubmed |
| pubmed-article:15927991 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15927991 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:15927991 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:15927991 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:15927991 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15927991 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15927991 | pubmed:month | May | lld:pubmed |
| pubmed-article:15927991 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:15927991 | pubmed:author | pubmed-author:EddahibiSaadi... | lld:pubmed |
| pubmed-article:15927991 | pubmed:author | pubmed-author:AdnotSergeS | lld:pubmed |
| pubmed-article:15927991 | pubmed:author | pubmed-author:HamonMichelM | lld:pubmed |
| pubmed-article:15927991 | pubmed:author | pubmed-author:RaffestinBern... | lld:pubmed |
| pubmed-article:15927991 | pubmed:author | pubmed-author:RaoulWilliamW | lld:pubmed |
| pubmed-article:15927991 | pubmed:author | pubmed-author:GuignabertChr... | lld:pubmed |
| pubmed-article:15927991 | pubmed:author | pubmed-author:RideauDominiq... | lld:pubmed |
| pubmed-article:15927991 | pubmed:author | pubmed-author:BenferhatRima... | lld:pubmed |
| pubmed-article:15927991 | pubmed:author | pubmed-author:ZadiguePatric... | lld:pubmed |
| pubmed-article:15927991 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:15927991 | pubmed:day | 31 | lld:pubmed |
| pubmed-article:15927991 | pubmed:volume | 111 | lld:pubmed |
| pubmed-article:15927991 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15927991 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15927991 | pubmed:pagination | 2812-9 | lld:pubmed |
| pubmed-article:15927991 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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| pubmed-article:15927991 | pubmed:meshHeading | pubmed-meshheading:15927991... | lld:pubmed |
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| pubmed-article:15927991 | pubmed:meshHeading | pubmed-meshheading:15927991... | lld:pubmed |
| pubmed-article:15927991 | pubmed:meshHeading | pubmed-meshheading:15927991... | lld:pubmed |
| pubmed-article:15927991 | pubmed:meshHeading | pubmed-meshheading:15927991... | lld:pubmed |
| pubmed-article:15927991 | pubmed:meshHeading | pubmed-meshheading:15927991... | lld:pubmed |
| pubmed-article:15927991 | pubmed:meshHeading | pubmed-meshheading:15927991... | lld:pubmed |
| pubmed-article:15927991 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:15927991 | pubmed:articleTitle | Serotonin transporter inhibition prevents and reverses monocrotaline-induced pulmonary hypertension in rats. | lld:pubmed |
| pubmed-article:15927991 | pubmed:affiliation | Département de Physiologie, INSERM U492, AP-HP, CHU Henri Mondor, Créteil, France. | lld:pubmed |
| pubmed-article:15927991 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15927991 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:15927991 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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