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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
5
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pubmed:dateCreated |
2005-5-11
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pubmed:abstractText |
To study the necessity for activating Fcgamma receptor types I and III (FcgammaRI and FcgammaRIII) in proteoglycan-induced arthritis (PGIA), a murine model of rheumatoid arthritis, and to determine whether usage of FcgammaRI or FcgammaRIII correlates with the Th1 phenotype or the autoantibody isotype in PGIA.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
May
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pubmed:issn |
0004-3591
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
52
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1612-9
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:15880819-Animals,
pubmed-meshheading:15880819-Arthritis,
pubmed-meshheading:15880819-Chemokines,
pubmed-meshheading:15880819-Cytokines,
pubmed-meshheading:15880819-Female,
pubmed-meshheading:15880819-Humans,
pubmed-meshheading:15880819-Mice,
pubmed-meshheading:15880819-Mice, Inbred BALB C,
pubmed-meshheading:15880819-Mice, SCID,
pubmed-meshheading:15880819-Proteoglycans,
pubmed-meshheading:15880819-RNA, Messenger,
pubmed-meshheading:15880819-Receptors, IgG,
pubmed-meshheading:15880819-T-Lymphocytes
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pubmed:year |
2005
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pubmed:articleTitle |
Development of proteoglycan-induced arthritis is critically dependent on Fcgamma receptor type III expression.
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pubmed:affiliation |
Rush University Medical Center, Chicago, Illinois, USA.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, N.I.H., Extramural
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