| pubmed-article:15880051 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:15880051 | lifeskim:mentions | umls-concept:C1709854 | lld:lifeskim |
| pubmed-article:15880051 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:15880051 | lifeskim:mentions | umls-concept:C0025919 | lld:lifeskim |
| pubmed-article:15880051 | lifeskim:mentions | umls-concept:C0085243 | lld:lifeskim |
| pubmed-article:15880051 | lifeskim:mentions | umls-concept:C0450127 | lld:lifeskim |
| pubmed-article:15880051 | lifeskim:mentions | umls-concept:C0079189 | lld:lifeskim |
| pubmed-article:15880051 | lifeskim:mentions | umls-concept:C0751781 | lld:lifeskim |
| pubmed-article:15880051 | lifeskim:mentions | umls-concept:C1261381 | lld:lifeskim |
| pubmed-article:15880051 | lifeskim:mentions | umls-concept:C0205307 | lld:lifeskim |
| pubmed-article:15880051 | lifeskim:mentions | umls-concept:C1426208 | lld:lifeskim |
| pubmed-article:15880051 | lifeskim:mentions | umls-concept:C1705241 | lld:lifeskim |
| pubmed-article:15880051 | lifeskim:mentions | umls-concept:C1705242 | lld:lifeskim |
| pubmed-article:15880051 | lifeskim:mentions | umls-concept:C2003905 | lld:lifeskim |
| pubmed-article:15880051 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:15880051 | pubmed:dateCreated | 2005-5-9 | lld:pubmed |
| pubmed-article:15880051 | pubmed:abstractText | The ability to block interferon signaling represents an important strategy in designing therapies to prevent beta-cell destruction during islet allograft rejection. | lld:pubmed |
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| pubmed-article:15880051 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:15880051 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:15880051 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15880051 | pubmed:month | May | lld:pubmed |
| pubmed-article:15880051 | pubmed:issn | 0041-1337 | lld:pubmed |
| pubmed-article:15880051 | pubmed:author | pubmed-author:SarvetnickNor... | lld:pubmed |
| pubmed-article:15880051 | pubmed:author | pubmed-author:Flodström-Tul... | lld:pubmed |
| pubmed-article:15880051 | pubmed:author | pubmed-author:SolomonMichel... | lld:pubmed |
| pubmed-article:15880051 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:15880051 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:15880051 | pubmed:volume | 79 | lld:pubmed |
| pubmed-article:15880051 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15880051 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15880051 | pubmed:pagination | 1104-9 | lld:pubmed |
| pubmed-article:15880051 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:15880051 | pubmed:meshHeading | pubmed-meshheading:15880051... | lld:pubmed |
| pubmed-article:15880051 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:15880051 | pubmed:articleTitle | Differences in suppressor of cytokine signaling-1 (SOCS-1) expressing islet allograft destruction in normal BALB/c and spontaneously-diabetic NOD recipient mice. | lld:pubmed |
| pubmed-article:15880051 | pubmed:affiliation | Department of Immunology, The Scripps Research Institute, La Jolla, CA 92037, USA. | lld:pubmed |
| pubmed-article:15880051 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15880051 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:15880051 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:15880051 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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