Source:http://linkedlifedata.com/resource/pubmed/id/15877800
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| Predicate | Object |
|---|---|
| rdf:type | |
| lifeskim:mentions | |
| pubmed:issue |
3
|
| pubmed:dateCreated |
2005-5-9
|
| pubmed:abstractText |
Cardiac allograft vasculopathy (CAV) limits survival after heart transplantation (HTx). Between immunologic and non-immunologic factors, reactive oxygen species generation has been proposed as pathogenetic mechanism. This study was aimed at evaluating redox status in HTx recipients and verifying whether it could be independently associated with CAV.
|
| pubmed:language |
eng
|
| pubmed:journal | |
| pubmed:citationSubset |
IM
|
| pubmed:chemical | |
| pubmed:status |
MEDLINE
|
| pubmed:month |
Jun
|
| pubmed:issn |
0902-0063
|
| pubmed:author | |
| pubmed:issnType |
Print
|
| pubmed:volume |
19
|
| pubmed:owner |
NLM
|
| pubmed:authorsComplete |
Y
|
| pubmed:pagination |
367-71
|
| pubmed:meshHeading |
pubmed-meshheading:15877800-Biological Markers,
pubmed-meshheading:15877800-Glutathione,
pubmed-meshheading:15877800-Heart Transplantation,
pubmed-meshheading:15877800-Humans,
pubmed-meshheading:15877800-Male,
pubmed-meshheading:15877800-Oxidation-Reduction,
pubmed-meshheading:15877800-Transplantation, Homologous,
pubmed-meshheading:15877800-Vascular Diseases
|
| pubmed:year |
2005
|
| pubmed:articleTitle |
Blood glutathione as a marker of cardiac allograft vasculopathy in heart transplant recipients.
|
| pubmed:affiliation |
CNR Clinical Physiology Institute, Section of Milan, Milan, Italy.
|
| pubmed:publicationType |
Journal Article
|