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| pubmed-article:15854903 | lifeskim:mentions | umls-concept:C0017280 | lld:lifeskim |
| pubmed-article:15854903 | lifeskim:mentions | umls-concept:C0038826 | lld:lifeskim |
| pubmed-article:15854903 | lifeskim:mentions | umls-concept:C0020964 | lld:lifeskim |
| pubmed-article:15854903 | lifeskim:mentions | umls-concept:C1332700 | lld:lifeskim |
| pubmed-article:15854903 | lifeskim:mentions | umls-concept:C1332714 | lld:lifeskim |
| pubmed-article:15854903 | lifeskim:mentions | umls-concept:C0679199 | lld:lifeskim |
| pubmed-article:15854903 | lifeskim:mentions | umls-concept:C0443211 | lld:lifeskim |
| pubmed-article:15854903 | lifeskim:mentions | umls-concept:C1554184 | lld:lifeskim |
| pubmed-article:15854903 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:15854903 | pubmed:dateCreated | 2005-4-27 | lld:pubmed |
| pubmed-article:15854903 | pubmed:abstractText | Viruses frequently render cells refractory to subsequent infection with the same virus. This state of superinfection immunity counteracts potentially detrimental consequences for the infected cell and facilitates high-level replication and viral spread in the host. | lld:pubmed |
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| pubmed-article:15854903 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:15854903 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:15854903 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15854903 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:15854903 | pubmed:issn | 0960-9822 | lld:pubmed |
| pubmed-article:15854903 | pubmed:author | pubmed-author:KepplerOliver... | lld:pubmed |
| pubmed-article:15854903 | pubmed:author | pubmed-author:MichelNicoN | lld:pubmed |
| pubmed-article:15854903 | pubmed:author | pubmed-author:FacklerOliver... | lld:pubmed |
| pubmed-article:15854903 | pubmed:author | pubmed-author:AllespachInaI | lld:pubmed |
| pubmed-article:15854903 | pubmed:author | pubmed-author:VenzkeStephan... | lld:pubmed |
| pubmed-article:15854903 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:15854903 | pubmed:day | 26 | lld:pubmed |
| pubmed-article:15854903 | pubmed:volume | 15 | lld:pubmed |
| pubmed-article:15854903 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15854903 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15854903 | pubmed:pagination | 714-23 | lld:pubmed |
| pubmed-article:15854903 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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| pubmed-article:15854903 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:15854903 | pubmed:articleTitle | The Nef protein of human immunodeficiency virus establishes superinfection immunity by a dual strategy to downregulate cell-surface CCR5 and CD4. | lld:pubmed |
| pubmed-article:15854903 | pubmed:affiliation | Department of Virology, University of Heidelberg, Germany. | lld:pubmed |
| pubmed-article:15854903 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15854903 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:15854903 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:15854903 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:15854903 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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