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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
4
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pubmed:dateCreated |
2005-4-11
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pubmed:abstractText |
The absence of all Kell blood group antigens (K(0) phenotype) is very rare. K(0) persons, however, can produce clinically significant anti-Ku (K5) after transfusion and/or pregnancy and require K(0) blood for transfusion. Ten alleles giving rise to the K(0) phenotype have been reported: different populations were studied although none from Scandinavia.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Apr
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pubmed:issn |
0041-1132
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
45
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
545-9
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:15819675-Alleles,
pubmed-meshheading:15819675-Alternative Splicing,
pubmed-meshheading:15819675-Codon, Nonsense,
pubmed-meshheading:15819675-Family Health,
pubmed-meshheading:15819675-Female,
pubmed-meshheading:15819675-Genetics, Population,
pubmed-meshheading:15819675-Humans,
pubmed-meshheading:15819675-Introns,
pubmed-meshheading:15819675-Kell Blood-Group System,
pubmed-meshheading:15819675-Male,
pubmed-meshheading:15819675-Pedigree,
pubmed-meshheading:15819675-Phenotype,
pubmed-meshheading:15819675-Point Mutation,
pubmed-meshheading:15819675-Sweden
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pubmed:year |
2005
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pubmed:articleTitle |
Genetic basis of the K(0) phenotype in the Swedish population.
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pubmed:affiliation |
Blood Center, University Hospital, Lund, Sweden.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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