Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
4
pubmed:dateCreated
2005-4-8
pubmed:abstractText
In Graves' disease (GD), stimulating anti-TSH receptor antibodies are responsible for hyperthyroidism. T-helper 2 (Th2) cells were expected to be involved in the underlying immune mechanism, although this is still controversial. The aim of this study was to examine the expression of CXCR6, a chemokine receptor that marks functionally specialized T-cells within the Th1 and T-cytotoxic 1 (Tc1) cell pool, to gain new insights into the running immune processes.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Apr
pubmed:issn
0804-4643
pubmed:author
pubmed:issnType
Print
pubmed:volume
152
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
635-43
pubmed:dateRevised
2006-11-15
pubmed:meshHeading
pubmed-meshheading:15817921-Adolescent, pubmed-meshheading:15817921-Adult, pubmed-meshheading:15817921-DNA, Complementary, pubmed-meshheading:15817921-Female, pubmed-meshheading:15817921-Flow Cytometry, pubmed-meshheading:15817921-Graves Disease, pubmed-meshheading:15817921-Humans, pubmed-meshheading:15817921-Killer Cells, Natural, pubmed-meshheading:15817921-Male, pubmed-meshheading:15817921-Middle Aged, pubmed-meshheading:15817921-Polymerase Chain Reaction, pubmed-meshheading:15817921-RNA, Messenger, pubmed-meshheading:15817921-Receptors, Chemokine, pubmed-meshheading:15817921-Receptors, Cytokine, pubmed-meshheading:15817921-Receptors, G-Protein-Coupled, pubmed-meshheading:15817921-Receptors, Virus, pubmed-meshheading:15817921-T-Lymphocytes, pubmed-meshheading:15817921-T-Lymphocytes, Cytotoxic, pubmed-meshheading:15817921-Th1 Cells, pubmed-meshheading:15817921-Thyroid Gland
pubmed:year
2005
pubmed:articleTitle
CXCR6 within T-helper (Th) and T-cytotoxic (Tc) type 1 lymphocytes in Graves' disease (GD).
pubmed:affiliation
Institute of Anatomy, University of Leipzig, Germany. ausg@medizin.uni-leipzig.de
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't