| pubmed-article:15809371 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:15809371 | lifeskim:mentions | umls-concept:C0012634 | lld:lifeskim |
| pubmed-article:15809371 | lifeskim:mentions | umls-concept:C0025914 | lld:lifeskim |
| pubmed-article:15809371 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:15809371 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
| pubmed-article:15809371 | lifeskim:mentions | umls-concept:C0439660 | lld:lifeskim |
| pubmed-article:15809371 | lifeskim:mentions | umls-concept:C0151654 | lld:lifeskim |
| pubmed-article:15809371 | lifeskim:mentions | umls-concept:C0457405 | lld:lifeskim |
| pubmed-article:15809371 | pubmed:issue | 14 | lld:pubmed |
| pubmed-article:15809371 | pubmed:dateCreated | 2005-4-12 | lld:pubmed |
| pubmed-article:15809371 | pubmed:abstractText | We have previously linked hereditary progressive cardiac conduction defect (hereditary Lenègre's disease) to a loss-of-function mutation in the gene encoding the main cardiac Na+ channel, SCN5A. In the present study, we investigated heterozygous Scn5a-knockout mice (Scn5a+/- mice) as a model for hereditary Lenègre's disease. | lld:pubmed |
| pubmed-article:15809371 | pubmed:language | eng | lld:pubmed |
| pubmed-article:15809371 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15809371 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:15809371 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:15809371 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15809371 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15809371 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:15809371 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:15809371 | pubmed:author | pubmed-author:GraceAndrew... | lld:pubmed |
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| pubmed-article:15809371 | pubmed:author | pubmed-author:GoddardCathar... | lld:pubmed |
| pubmed-article:15809371 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:15809371 | pubmed:day | 12 | lld:pubmed |
| pubmed-article:15809371 | pubmed:volume | 111 | lld:pubmed |
| pubmed-article:15809371 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15809371 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15809371 | pubmed:pagination | 1738-46 | lld:pubmed |
| pubmed-article:15809371 | pubmed:dateRevised | 2011-7-22 | lld:pubmed |
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| pubmed-article:15809371 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:15809371 | pubmed:articleTitle | Mouse model of SCN5A-linked hereditary Lenègre's disease: age-related conduction slowing and myocardial fibrosis. | lld:pubmed |
| pubmed-article:15809371 | pubmed:affiliation | INSERM U533, Institut du Thorax, Faculté de Médecine, Nantes, France. | lld:pubmed |
| pubmed-article:15809371 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15809371 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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