| pubmed-article:15795102 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:15795102 | lifeskim:mentions | umls-concept:C0684249 | lld:lifeskim |
| pubmed-article:15795102 | lifeskim:mentions | umls-concept:C0035143 | lld:lifeskim |
| pubmed-article:15795102 | lifeskim:mentions | umls-concept:C0079904 | lld:lifeskim |
| pubmed-article:15795102 | lifeskim:mentions | umls-concept:C0021467 | lld:lifeskim |
| pubmed-article:15795102 | lifeskim:mentions | umls-concept:C2699153 | lld:lifeskim |
| pubmed-article:15795102 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
| pubmed-article:15795102 | lifeskim:mentions | umls-concept:C0475264 | lld:lifeskim |
| pubmed-article:15795102 | lifeskim:mentions | umls-concept:C0021469 | lld:lifeskim |
| pubmed-article:15795102 | lifeskim:mentions | umls-concept:C0059438 | lld:lifeskim |
| pubmed-article:15795102 | lifeskim:mentions | umls-concept:C1314939 | lld:lifeskim |
| pubmed-article:15795102 | lifeskim:mentions | umls-concept:C2911684 | lld:lifeskim |
| pubmed-article:15795102 | lifeskim:mentions | umls-concept:C0185117 | lld:lifeskim |
| pubmed-article:15795102 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:15795102 | pubmed:dateCreated | 2005-3-29 | lld:pubmed |
| pubmed-article:15795102 | pubmed:abstractText | Epigallocatechin-3-gallate (EGCG) repressed the invasion of lung carcinoma 95-D cells in invasion assay. RT-PCR analysis illuminated that 40 microM EGCG down-regulated the expression of MMP-9 by 45.7% and the result of Western blot analysis provided further evidence. NF-kappa B localized in the nucleus of the 95-D cells was diminished in a dose-dependent manner in EGCG-treated cells as shown by Western blot. Intracellular oxidants were more abundant in invasive cells than in invasion-suppressed cells fed with EGCG for 18 h. Thus, the inhibition of tumor invasion by EGCG was shown to be attributed to decreases of the expression of MMP-9 and NF-kappa B, which may result from decrease of intracellular oxidants. | lld:pubmed |
| pubmed-article:15795102 | pubmed:language | eng | lld:pubmed |
| pubmed-article:15795102 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15795102 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:15795102 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15795102 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15795102 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15795102 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15795102 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15795102 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15795102 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15795102 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:15795102 | pubmed:issn | 0753-3322 | lld:pubmed |
| pubmed-article:15795102 | pubmed:author | pubmed-author:LiuJianwenJ | lld:pubmed |
| pubmed-article:15795102 | pubmed:author | pubmed-author:WeiDongzhiD | lld:pubmed |
| pubmed-article:15795102 | pubmed:author | pubmed-author:YangJingyaJ | lld:pubmed |
| pubmed-article:15795102 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:15795102 | pubmed:volume | 59 | lld:pubmed |
| pubmed-article:15795102 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15795102 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15795102 | pubmed:pagination | 98-103 | lld:pubmed |
| pubmed-article:15795102 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:15795102 | pubmed:meshHeading | pubmed-meshheading:15795102... | lld:pubmed |
| pubmed-article:15795102 | pubmed:meshHeading | pubmed-meshheading:15795102... | lld:pubmed |
| pubmed-article:15795102 | pubmed:meshHeading | pubmed-meshheading:15795102... | lld:pubmed |
| pubmed-article:15795102 | pubmed:meshHeading | pubmed-meshheading:15795102... | lld:pubmed |
| pubmed-article:15795102 | pubmed:meshHeading | pubmed-meshheading:15795102... | lld:pubmed |
| pubmed-article:15795102 | pubmed:meshHeading | pubmed-meshheading:15795102... | lld:pubmed |
| pubmed-article:15795102 | pubmed:meshHeading | pubmed-meshheading:15795102... | lld:pubmed |
| pubmed-article:15795102 | pubmed:meshHeading | pubmed-meshheading:15795102... | lld:pubmed |
| pubmed-article:15795102 | pubmed:meshHeading | pubmed-meshheading:15795102... | lld:pubmed |
| pubmed-article:15795102 | pubmed:meshHeading | pubmed-meshheading:15795102... | lld:pubmed |
| pubmed-article:15795102 | pubmed:meshHeading | pubmed-meshheading:15795102... | lld:pubmed |
| pubmed-article:15795102 | pubmed:meshHeading | pubmed-meshheading:15795102... | lld:pubmed |
| pubmed-article:15795102 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:15795102 | pubmed:articleTitle | Repressions of MMP-9 expression and NF-kappa B localization are involved in inhibition of lung carcinoma 95-D cell invasion by (-)-epigallocatechin-3-gallate. | lld:pubmed |
| pubmed-article:15795102 | pubmed:affiliation | State Key Laboratory of Bioreactor Engineering, New World Institute of Biotechnology, East China University of Science and Technology, Shanghai 200237, PR China. | lld:pubmed |
| pubmed-article:15795102 | pubmed:publicationType | Journal Article | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:15795102 | lld:pubmed |
