pubmed-article:15769913 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:15769913 | lifeskim:mentions | umls-concept:C0003873 | lld:lifeskim |
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pubmed-article:15769913 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
pubmed-article:15769913 | lifeskim:mentions | umls-concept:C0205216 | lld:lifeskim |
pubmed-article:15769913 | lifeskim:mentions | umls-concept:C0666743 | lld:lifeskim |
pubmed-article:15769913 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
pubmed-article:15769913 | lifeskim:mentions | umls-concept:C0205210 | lld:lifeskim |
pubmed-article:15769913 | lifeskim:mentions | umls-concept:C0205269 | lld:lifeskim |
pubmed-article:15769913 | lifeskim:mentions | umls-concept:C2911684 | lld:lifeskim |
pubmed-article:15769913 | lifeskim:mentions | umls-concept:C0242708 | lld:lifeskim |
pubmed-article:15769913 | lifeskim:mentions | umls-concept:C0185117 | lld:lifeskim |
pubmed-article:15769913 | pubmed:issue | 4 | lld:pubmed |
pubmed-article:15769913 | pubmed:dateCreated | 2005-3-16 | lld:pubmed |
pubmed-article:15769913 | pubmed:abstractText | Tumour necrosis alpha (TNF alpha) blocking agents lead to pronounced clinical effects and reduced synovial infiltrate in rheumatoid arthritis. Laboratory and clinical studies suggest that TNF alpha independent pathways play a role in the disease. | lld:pubmed |
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pubmed-article:15769913 | pubmed:language | eng | lld:pubmed |
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pubmed-article:15769913 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:15769913 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:15769913 | pubmed:month | Apr | lld:pubmed |
pubmed-article:15769913 | pubmed:issn | 0003-4967 | lld:pubmed |
pubmed-article:15769913 | pubmed:author | pubmed-author:ShubT ATA | lld:pubmed |
pubmed-article:15769913 | pubmed:author | pubmed-author:van LaarJ MJM | lld:pubmed |
pubmed-article:15769913 | pubmed:author | pubmed-author:van... | lld:pubmed |
pubmed-article:15769913 | pubmed:author | pubmed-author:HuizingaT W... | lld:pubmed |
pubmed-article:15769913 | pubmed:author | pubmed-author:ToesR E MRE | lld:pubmed |
pubmed-article:15769913 | pubmed:author | pubmed-author:LevarhtE W... | lld:pubmed |
pubmed-article:15769913 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:15769913 | pubmed:volume | 64 | lld:pubmed |
pubmed-article:15769913 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:15769913 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:15769913 | pubmed:pagination | 537-43 | lld:pubmed |
pubmed-article:15769913 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:15769913 | pubmed:year | 2005 | lld:pubmed |
pubmed-article:15769913 | pubmed:articleTitle | Clinical efficacy of infliximab plus methotrexate in DMARD naive and DMARD refractory rheumatoid arthritis is associated with decreased synovial expression of TNF alpha and IL18 but not CXCL12. | lld:pubmed |
pubmed-article:15769913 | pubmed:affiliation | Leiden University Medical Centre, Department of Rheumatology, PO Box 9600, 2300 RC Leiden, Netherlands. M.van_Oosterhout@LUMC.nl | lld:pubmed |
pubmed-article:15769913 | pubmed:publicationType | Journal Article | lld:pubmed |