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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
4
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pubmed:dateCreated |
2005-7-27
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pubmed:abstractText |
One of the difficulties confronting genetic studies of prostate cancer is the complex and heterogeneous etiology. Given the high population frequency of lesions meeting the histological definition of prostate cancer, a significant portion of men with a positive family history may be diagnosed due to increased surveillance and associated higher likelihood of biopsy. Over diagnosis decreases power to detect genes that increase susceptibility to a clinically significant prostate cancer.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:status |
MEDLINE
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pubmed:month |
Sep
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pubmed:issn |
0270-4137
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pubmed:author |
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pubmed:copyrightInfo |
Copyright 2005 Wiley-Liss, Inc.
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pubmed:issnType |
Print
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pubmed:day |
1
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pubmed:volume |
64
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
356-61
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pubmed:dateRevised |
2010-11-18
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pubmed:meshHeading |
pubmed-meshheading:15754351-Family Health,
pubmed-meshheading:15754351-Genetic Linkage,
pubmed-meshheading:15754351-Genetic Predisposition to Disease,
pubmed-meshheading:15754351-Genetic Testing,
pubmed-meshheading:15754351-Genome, Human,
pubmed-meshheading:15754351-Humans,
pubmed-meshheading:15754351-Male,
pubmed-meshheading:15754351-Prostatic Neoplasms,
pubmed-meshheading:15754351-Risk Factors,
pubmed-meshheading:15754351-Severity of Illness Index
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pubmed:year |
2005
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pubmed:articleTitle |
Genome-wide screen for prostate cancer susceptibility genes in men with clinically significant disease.
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pubmed:affiliation |
Center for Human Genomics, Wake Forest University School of Medicine, Winston-Salem, North Carolina 27157, USA.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't,
Research Support, N.I.H., Extramural
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