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pubmed-article:15746989pubmed:abstractTextThe functional status and mechanism of increased VDR in GHS rats were investigated. Basal VDR and calbindins were increased in GHS rats. 1,25(OH)(2)D(3) increased VDR and calbindins in controls but not GHS rats. VDR half-life was prolonged in GHS rats. This study supports the mechanism and functional status of elevated VDR in GHS rats.lld:pubmed
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pubmed-article:15746989pubmed:articleTitleMechanism and function of high vitamin D receptor levels in genetic hypercalciuric stone-forming rats.lld:pubmed
pubmed-article:15746989pubmed:affiliationDepartment of Medicine, University of Chicago Pritzker School of Medicine, Chicago, Illinois 60637, USA.lld:pubmed
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pubmed-article:15746989pubmed:publicationTypeResearch Support, U.S. Gov't, P.H.S.lld:pubmed
pubmed-article:15746989pubmed:publicationTypeResearch Support, N.I.H., Extramurallld:pubmed
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