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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
3
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pubmed:dateCreated |
2005-3-4
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pubmed:abstractText |
The functional status and mechanism of increased VDR in GHS rats were investigated. Basal VDR and calbindins were increased in GHS rats. 1,25(OH)(2)D(3) increased VDR and calbindins in controls but not GHS rats. VDR half-life was prolonged in GHS rats. This study supports the mechanism and functional status of elevated VDR in GHS rats.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
|
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pubmed:status |
MEDLINE
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pubmed:month |
Mar
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pubmed:issn |
0884-0431
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:volume |
20
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
447-54
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:15746989-Animals,
pubmed-meshheading:15746989-Biological Transport,
pubmed-meshheading:15746989-Calcitriol,
pubmed-meshheading:15746989-Calcium Channel Agonists,
pubmed-meshheading:15746989-Calcium-Binding Protein, Vitamin D-Dependent,
pubmed-meshheading:15746989-Duodenum,
pubmed-meshheading:15746989-Gene Expression Regulation,
pubmed-meshheading:15746989-Hypercalcemia,
pubmed-meshheading:15746989-Kidney,
pubmed-meshheading:15746989-Male,
pubmed-meshheading:15746989-Rats,
pubmed-meshheading:15746989-Receptors, Calcitriol,
pubmed-meshheading:15746989-Urinary Calculi
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pubmed:year |
2005
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pubmed:articleTitle |
Mechanism and function of high vitamin D receptor levels in genetic hypercalciuric stone-forming rats.
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pubmed:affiliation |
Department of Medicine, University of Chicago Pritzker School of Medicine, Chicago, Illinois 60637, USA.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, N.I.H., Extramural
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