Source:http://linkedlifedata.com/resource/pubmed/id/15703605
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| Predicate | Object |
|---|---|
| rdf:type | |
| lifeskim:mentions | |
| pubmed:issue |
6
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| pubmed:dateCreated |
2005-2-10
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| pubmed:abstractText |
In response to the presence of pathogens in the environment, the innate immune system has evolved to provide a rapid defense against microbes. This response involves the recognition of molecular patterns present in diverse microbes by a series of receptors termed toll-like receptors. The focus of this article is the regulation of toll-like receptor signaling in the intestinal epithelium. The intestinal epithelium is continually exposed to a high concentration of diverse bacteria. In spite of the density of commensal bacteria, the normal intestine is not inflamed. Idiopathic inflammatory bowel disease in humans and animals is characterized by aberrant host-microbial interactions.
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| pubmed:language |
eng
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| pubmed:journal | |
| pubmed:status |
PubMed-not-MEDLINE
|
| pubmed:month |
Nov
|
| pubmed:issn |
0267-1379
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| pubmed:author | |
| pubmed:issnType |
Print
|
| pubmed:volume |
19
|
| pubmed:owner |
NLM
|
| pubmed:authorsComplete |
Y
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| pubmed:pagination |
559-64
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| pubmed:dateRevised |
2006-5-9
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| pubmed:year |
2003
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| pubmed:articleTitle |
Immunologic regulation of toll-like receptors in gut epithelium.
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| pubmed:affiliation |
Inflammatory Bowel Disease Center, Division of Gastroenterology, Department of Medicine, Burns and Allen Research Institute, Cedars-Sinai Medical Center, Los Angeles, California 90048, USA. maria.abreu@cshs.org
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| pubmed:publicationType |
Journal Article
|