Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
5
pubmed:dateCreated
2005-2-8
pubmed:abstractText
S100B, a 20-kDa, Ca2+-binding dimer, is a putative intrinsic negative regulator of myocardial hypertrophy expressed after myocardial infarction. S100B-overexpressing transgenic (TG) and S100B-knockout (KO) mice have been generated to assess the consequences of S100B expression and altered hypertrophy after infarction.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Feb
pubmed:issn
1524-4539
pubmed:author
pubmed:issnType
Electronic
pubmed:day
8
pubmed:volume
111
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
598-606
pubmed:dateRevised
2006-11-15
pubmed:meshHeading
pubmed-meshheading:15699280-Animals, pubmed-meshheading:15699280-Apoptosis, pubmed-meshheading:15699280-Gene Expression, pubmed-meshheading:15699280-Heart, pubmed-meshheading:15699280-Heart Ventricles, pubmed-meshheading:15699280-Hypertrophy, Left Ventricular, pubmed-meshheading:15699280-Mice, pubmed-meshheading:15699280-Mice, Knockout, pubmed-meshheading:15699280-Mice, Transgenic, pubmed-meshheading:15699280-Myocardial Infarction, pubmed-meshheading:15699280-Myocardium, pubmed-meshheading:15699280-Myocytes, Cardiac, pubmed-meshheading:15699280-Nerve Growth Factors, pubmed-meshheading:15699280-Protein Biosynthesis, pubmed-meshheading:15699280-S100 Proteins, pubmed-meshheading:15699280-Survival Analysis, pubmed-meshheading:15699280-Ventricular Function, Left, pubmed-meshheading:15699280-Ventricular Remodeling
pubmed:year
2005
pubmed:articleTitle
S100B expression modulates left ventricular remodeling after myocardial infarction in mice.
pubmed:affiliation
Division of Cardiology, St Michael's Hospital, Toronto, Ontario, Canada.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't