Linked Life Data

15692785

Source:http://linkedlifedata.com/resource/pubmed/id/15692785

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
2005-2-4
pubmed:abstractText
Gut commensal microbes affect the development and activation of the mucosal and systemic immune systems. However, the exact molecular mechanism of these microbes that is involved in the development of colitis remains unclear.
pubmed:commentsCorrections
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
http://linkedlifedata.com/resource/pubmed/chemical/Adaptor Proteins, Signal Transducing, http://linkedlifedata.com/resource/pubmed/chemical/Antigens, CD, http://linkedlifedata.com/resource/pubmed/chemical/Antigens, Differentiation, http://linkedlifedata.com/resource/pubmed/chemical/Antigens, Differentiation..., http://linkedlifedata.com/resource/pubmed/chemical/Antiviral Agents, http://linkedlifedata.com/resource/pubmed/chemical/CD69 antigen, http://linkedlifedata.com/resource/pubmed/chemical/Dextran Sulfate, http://linkedlifedata.com/resource/pubmed/chemical/Il18r1 protein, mouse, http://linkedlifedata.com/resource/pubmed/chemical/Interleukin-18 Receptor alpha..., http://linkedlifedata.com/resource/pubmed/chemical/Lectins, C-Type, http://linkedlifedata.com/resource/pubmed/chemical/Myd88 protein, mouse, http://linkedlifedata.com/resource/pubmed/chemical/Myeloid Differentiation Factor 88, http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Immunologic, http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Interleukin, http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Interleukin-18, http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Interleukin-2
pubmed:status
MEDLINE
pubmed:month
Jan
pubmed:issn
0944-1174
pubmed:author
pubmed:issnType
Print
pubmed:volume
40
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
16-23
pubmed:dateRevised
2010-11-18
pubmed:meshHeading
pubmed-meshheading:15692785-Adaptor Proteins, Signal Transducing, pubmed-meshheading:15692785-Animals, pubmed-meshheading:15692785-Antigens, CD, pubmed-meshheading:15692785-Antigens, Differentiation, pubmed-meshheading:15692785-Antigens, Differentiation, T-Lymphocyte, pubmed-meshheading:15692785-Antiviral Agents, pubmed-meshheading:15692785-CD4-Positive T-Lymphocytes, pubmed-meshheading:15692785-Colitis, pubmed-meshheading:15692785-Dextran Sulfate, pubmed-meshheading:15692785-Disease Models, Animal, pubmed-meshheading:15692785-Disease Susceptibility, pubmed-meshheading:15692785-Dose-Response Relationship, Drug, pubmed-meshheading:15692785-Immunohistochemistry, pubmed-meshheading:15692785-Inflammatory Bowel Diseases, pubmed-meshheading:15692785-Interleukin-18 Receptor alpha Subunit, pubmed-meshheading:15692785-Intestinal Mucosa, pubmed-meshheading:15692785-Lectins, C-Type, pubmed-meshheading:15692785-Macrophages, pubmed-meshheading:15692785-Male, pubmed-meshheading:15692785-Mice, pubmed-meshheading:15692785-Myeloid Differentiation Factor 88, pubmed-meshheading:15692785-Receptors, Immunologic, pubmed-meshheading:15692785-Receptors, Interleukin, pubmed-meshheading:15692785-Receptors, Interleukin-18, pubmed-meshheading:15692785-Receptors, Interleukin-2, pubmed-meshheading:15692785-Severity of Illness Index
pubmed:year
2005
pubmed:articleTitle
MyD88-deficient mice develop severe intestinal inflammation in dextran sodium sulfate colitis.
pubmed:affiliation
Department of Gastroenterology and Hepatology, Graduate School, Tokyo Medical and Dental University, 1-5-45 Yushima, Bunkyo-ku, Tokyo 113-8519, Japan.
pubmed:publicationType
Journal Article, Comparative Study, Research Support, Non-U.S. Gov't, Evaluation Studies