Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
2005-1-26
pubmed:abstractText
The tumor suppressor genes RASSF1A, APC, H-cadherin, RARbeta2, and cyclin D2 are methylated more frequently in breast cancer than in adjacent benign tissue. However, it is unclear whether promoter methylation of tumor suppressor genes in benign breast tissue is associated with an increased risk for breast cancer.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jan
pubmed:issn
1078-0432
pubmed:author
pubmed:issnType
Print
pubmed:day
1
pubmed:volume
11
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
166-72
pubmed:dateRevised
2009-11-19
pubmed:meshHeading
pubmed-meshheading:15671542-Adult, pubmed-meshheading:15671542-Age Factors, pubmed-meshheading:15671542-Aged, pubmed-meshheading:15671542-Biopsy, pubmed-meshheading:15671542-Breast, pubmed-meshheading:15671542-Breast Neoplasms, pubmed-meshheading:15671542-Cadherins, pubmed-meshheading:15671542-Cyclin D2, pubmed-meshheading:15671542-Cyclins, pubmed-meshheading:15671542-DNA Methylation, pubmed-meshheading:15671542-Female, pubmed-meshheading:15671542-Humans, pubmed-meshheading:15671542-Middle Aged, pubmed-meshheading:15671542-Models, Theoretical, pubmed-meshheading:15671542-Polymerase Chain Reaction, pubmed-meshheading:15671542-Promoter Regions, Genetic, pubmed-meshheading:15671542-Protein C, pubmed-meshheading:15671542-Receptors, Retinoic Acid, pubmed-meshheading:15671542-Risk, pubmed-meshheading:15671542-Tumor Suppressor Proteins
pubmed:year
2005
pubmed:articleTitle
Promoter hypermethylation in benign breast epithelium in relation to predicted breast cancer risk.
pubmed:affiliation
Hamon Center for Therapeutic Oncology Research, Department of Surgery, University of Texas Southwestern Medical Center at Dallas, Dallas, Texas 75390-9155, USA.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, P.H.S., Research Support, Non-U.S. Gov't, Research Support, N.I.H., Extramural