| pubmed-article:15671286 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:15671286 | lifeskim:mentions | umls-concept:C0024660 | lld:lifeskim |
| pubmed-article:15671286 | lifeskim:mentions | umls-concept:C0010031 | lld:lifeskim |
| pubmed-article:15671286 | lifeskim:mentions | umls-concept:C0022568 | lld:lifeskim |
| pubmed-article:15671286 | lifeskim:mentions | umls-concept:C0670896 | lld:lifeskim |
| pubmed-article:15671286 | lifeskim:mentions | umls-concept:C1336636 | lld:lifeskim |
| pubmed-article:15671286 | lifeskim:mentions | umls-concept:C1423633 | lld:lifeskim |
| pubmed-article:15671286 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:15671286 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
| pubmed-article:15671286 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:15671286 | pubmed:dateCreated | 2005-1-26 | lld:pubmed |
| pubmed-article:15671286 | pubmed:abstractText | Toll-like receptors (TLRs), which recognize microbial products, have an important role in the host innate immune response. The purpose of the present study was to determine whether activation of these receptors leads to development of keratitis and to assess the role of the common adaptor molecule myeloid differentiation factor-88 (MyD88). | lld:pubmed |
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| pubmed-article:15671286 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:15671286 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:15671286 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15671286 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:15671286 | pubmed:issn | 0146-0404 | lld:pubmed |
| pubmed-article:15671286 | pubmed:author | pubmed-author:LUES CSC | lld:pubmed |
| pubmed-article:15671286 | pubmed:author | pubmed-author:HiseAmy GAG | lld:pubmed |
| pubmed-article:15671286 | pubmed:author | pubmed-author:LassJonathan... | lld:pubmed |
| pubmed-article:15671286 | pubmed:author | pubmed-author:DiaconuEugeni... | lld:pubmed |
| pubmed-article:15671286 | pubmed:author | pubmed-author:PearlmanEricE | lld:pubmed |
| pubmed-article:15671286 | pubmed:author | pubmed-author:GolenbockDoug... | lld:pubmed |
| pubmed-article:15671286 | pubmed:author | pubmed-author:HeinzelFred... | lld:pubmed |
| pubmed-article:15671286 | pubmed:author | pubmed-author:JohnsonAngela... | lld:pubmed |
| pubmed-article:15671286 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:15671286 | pubmed:volume | 46 | lld:pubmed |
| pubmed-article:15671286 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15671286 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15671286 | pubmed:pagination | 589-95 | lld:pubmed |
| pubmed-article:15671286 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:15671286 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:15671286 | pubmed:articleTitle | Activation of toll-like receptor (TLR)2, TLR4, and TLR9 in the mammalian cornea induces MyD88-dependent corneal inflammation. | lld:pubmed |
| pubmed-article:15671286 | pubmed:affiliation | Department of Ophthalmology, Case Western University, Cleveland, OH 44106-7286, USA. | lld:pubmed |
| pubmed-article:15671286 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15671286 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:15671286 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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