pubmed-article:15653992 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:15653992 | lifeskim:mentions | umls-concept:C0036202 | lld:lifeskim |
pubmed-article:15653992 | lifeskim:mentions | umls-concept:C0002390 | lld:lifeskim |
pubmed-article:15653992 | lifeskim:mentions | umls-concept:C0085236 | lld:lifeskim |
pubmed-article:15653992 | lifeskim:mentions | umls-concept:C0077503 | lld:lifeskim |
pubmed-article:15653992 | lifeskim:mentions | umls-concept:C1749467 | lld:lifeskim |
pubmed-article:15653992 | lifeskim:mentions | umls-concept:C1456820 | lld:lifeskim |
pubmed-article:15653992 | lifeskim:mentions | umls-concept:C0033268 | lld:lifeskim |
pubmed-article:15653992 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:15653992 | pubmed:dateCreated | 2005-1-17 | lld:pubmed |
pubmed-article:15653992 | pubmed:abstractText | Alveolar macrophage (AM)-derived tumor necrosis factor (TNF)-alpha plays a pivotal role in the pathogenesis of sarcoidosis and extrinsic allergic alveolitis (EAA). The effects of TNF-alpha are mediated by membrane TNF receptor (mTNFR)-1 and mTNFR-2, and can be blocked by soluble TNF receptor (sTNFR)-1 and sTNFR-2. | lld:pubmed |
pubmed-article:15653992 | pubmed:language | eng | lld:pubmed |
pubmed-article:15653992 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15653992 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:15653992 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15653992 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:15653992 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15653992 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:15653992 | pubmed:month | Jan | lld:pubmed |
pubmed-article:15653992 | pubmed:issn | 0012-3692 | lld:pubmed |
pubmed-article:15653992 | pubmed:author | pubmed-author:CostabelUlric... | lld:pubmed |
pubmed-article:15653992 | pubmed:author | pubmed-author:GuzmanJosuneJ | lld:pubmed |
pubmed-article:15653992 | pubmed:author | pubmed-author:DaiHuapingH | lld:pubmed |
pubmed-article:15653992 | pubmed:author | pubmed-author:ChenBaominB | lld:pubmed |
pubmed-article:15653992 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:15653992 | pubmed:volume | 127 | lld:pubmed |
pubmed-article:15653992 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:15653992 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:15653992 | pubmed:pagination | 251-6 | lld:pubmed |
pubmed-article:15653992 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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pubmed-article:15653992 | pubmed:meshHeading | pubmed-meshheading:15653992... | lld:pubmed |
pubmed-article:15653992 | pubmed:year | 2005 | lld:pubmed |
pubmed-article:15653992 | pubmed:articleTitle | Production of soluble tumor necrosis factor receptors and tumor necrosis factor-alpha by alveolar macrophages in sarcoidosis and extrinsic allergic alveolitis. | lld:pubmed |
pubmed-article:15653992 | pubmed:affiliation | Department of Penumology and Allergy, Ruhrlandklinik, Medical Faculty, University of Essen, Essen, Germany. | lld:pubmed |
pubmed-article:15653992 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:15653992 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
entrez-gene:24835 | entrezgene:pubmed | pubmed-article:15653992 | lld:entrezgene |
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