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rdf:type |
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lifeskim:mentions |
umls-concept:C0006560,
umls-concept:C0014257,
umls-concept:C0016017,
umls-concept:C0040044,
umls-concept:C0205227,
umls-concept:C0221099,
umls-concept:C0241669,
umls-concept:C0332281,
umls-concept:C0442805,
umls-concept:C0871261,
umls-concept:C1442488,
umls-concept:C1704632,
umls-concept:C1706817,
umls-concept:C1879547,
umls-concept:C2349975,
umls-concept:C2911692
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pubmed:issue |
1
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pubmed:dateCreated |
2005-1-4
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pubmed:abstractText |
The goal of this study was to determine whether chronic inflammation of the vascular wall may be associated with an impaired activation of the fibrinolytic system.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Jan
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pubmed:issn |
0735-1097
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pubmed:author |
pubmed-author:GeppertAlexanderA,
pubmed-author:HuberKurtK,
pubmed-author:JordanovaNelliN,
pubmed-author:MaurerGeraldG,
pubmed-author:NiessnerAlexanderA,
pubmed-author:NikfardjamMariamM,
pubmed-author:SchreiberWolfgangW,
pubmed-author:SpeidlWalter StefanWS,
pubmed-author:WojtaJohannJ,
pubmed-author:ZeinerAndreaA,
pubmed-author:ZornGerlindeG
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pubmed:issnType |
Print
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pubmed:day |
4
|
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pubmed:volume |
45
|
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
|
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pubmed:pagination |
30-4
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pubmed:dateRevised |
2007-11-15
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pubmed:meshHeading |
pubmed-meshheading:15629369-Aged,
pubmed-meshheading:15629369-C-Reactive Protein,
pubmed-meshheading:15629369-Coronary Artery Disease,
pubmed-meshheading:15629369-Endothelium, Vascular,
pubmed-meshheading:15629369-Female,
pubmed-meshheading:15629369-Fibrinolysis,
pubmed-meshheading:15629369-Humans,
pubmed-meshheading:15629369-Male,
pubmed-meshheading:15629369-Middle Aged,
pubmed-meshheading:15629369-Multivariate Analysis,
pubmed-meshheading:15629369-Myocardial Infarction,
pubmed-meshheading:15629369-Plasminogen Activator Inhibitor 1,
pubmed-meshheading:15629369-Tissue Plasminogen Activator
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pubmed:year |
2005
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pubmed:articleTitle |
An increase of C-reactive protein is associated with enhanced activation of endogenous fibrinolysis at baseline but an impaired endothelial fibrinolytic response after venous occlusion.
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pubmed:affiliation |
Department of Internal Medicine II, Medical University of Vienna and the Ludwig Boltzmann Foundation for Cardiovascular Research, Vienna, Austria.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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