Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
3
pubmed:dateCreated
2005-2-15
pubmed:abstractText
Overexpression of the human transcription factor FOXC2 gene (FOXC2) protects against insulin resistance in mice and a common FOXC2 polymorphism (-512C>T) has been suggested to be associated with insulin resistance in humans. Here, we addressed the potential role for FOXC2 as a candidate gene for type 2 diabetes and associated phenotypes.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Mar
pubmed:issn
0307-0565
pubmed:author
pubmed:issnType
Print
pubmed:volume
29
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
268-74
pubmed:dateRevised
2009-9-15
pubmed:meshHeading
pubmed-meshheading:15597109-Aged, pubmed-meshheading:15597109-Body Mass Index, pubmed-meshheading:15597109-Case-Control Studies, pubmed-meshheading:15597109-DNA-Binding Proteins, pubmed-meshheading:15597109-Diabetes Mellitus, Type 2, pubmed-meshheading:15597109-Female, pubmed-meshheading:15597109-Forkhead Transcription Factors, pubmed-meshheading:15597109-Gene Frequency, pubmed-meshheading:15597109-Genetic Predisposition to Disease, pubmed-meshheading:15597109-Genotype, pubmed-meshheading:15597109-Humans, pubmed-meshheading:15597109-Insulin Resistance, pubmed-meshheading:15597109-Male, pubmed-meshheading:15597109-Metabolic Syndrome X, pubmed-meshheading:15597109-Middle Aged, pubmed-meshheading:15597109-Obesity, pubmed-meshheading:15597109-Phenotype, pubmed-meshheading:15597109-Polymorphism, Genetic, pubmed-meshheading:15597109-Transcription Factors
pubmed:year
2005
pubmed:articleTitle
Role of the FOXC2 -512C>T polymorphism in type 2 diabetes: possible association with the dysmetabolic syndrome.
pubmed:affiliation
Department of Endocrinology, Lund University, University Hospital MAS, Malmö, Sweden. emma.carlsson@endo.mas.lu.se
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't