Statements in which the resource exists.
SubjectPredicateObjectContext
pubmed-article:15591839rdf:typepubmed:Citationlld:pubmed
pubmed-article:15591839lifeskim:mentionsumls-concept:C0086418lld:lifeskim
pubmed-article:15591839lifeskim:mentionsumls-concept:C0027950lld:lifeskim
pubmed-article:15591839lifeskim:mentionsumls-concept:C0162638lld:lifeskim
pubmed-article:15591839lifeskim:mentionsumls-concept:C0302523lld:lifeskim
pubmed-article:15591839lifeskim:mentionsumls-concept:C0700325lld:lifeskim
pubmed-article:15591839lifeskim:mentionsumls-concept:C0014323lld:lifeskim
pubmed-article:15591839lifeskim:mentionsumls-concept:C1444748lld:lifeskim
pubmed-article:15591839pubmed:issue4lld:pubmed
pubmed-article:15591839pubmed:dateCreated2004-12-13lld:pubmed
pubmed-article:15591839pubmed:abstractTextNeutrophils are important effector cells against protozoan extracellular parasite Entamoeba histolytica, which causes amoebic colitis and liver abscess in human beings. Apoptotic cell death of neutrophils is an important event in the resolution of inflammation and parasite's survival in vivo. This study was undertaken to investigate the ultrastructural aspects of apoptotic cells during neutrophil death triggered by Entamoeba histolytica. Isolated human neutrophils from the peripheral blood were incubated with or without live trophozoites of E. histolytica and examined by transmission electron microscopy (TEM). Neutrophils incubated with E. histolytica were observed to show apoptotic characteristics, such as compaction of the nuclear chromatin and swelling of the nuclear envelop. In contrast, neutrophils incubated in the absence of the amoeba had many protrusions of irregular cell surfaces and heterogenous nuclear chromatin. Therefore, it is suggested that Entamoeba-induced neutrophil apoptosis contribute to prevent unwanted tissue inflammation and damage in the amoeba-invaded lesions in vivo.lld:pubmed
pubmed-article:15591839pubmed:commentsCorrectionshttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:15591839pubmed:commentsCorrectionshttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:15591839pubmed:commentsCorrectionshttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:15591839pubmed:commentsCorrectionshttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:15591839pubmed:commentsCorrectionshttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:15591839pubmed:commentsCorrectionshttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:15591839pubmed:commentsCorrectionshttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:15591839pubmed:commentsCorrectionshttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:15591839pubmed:commentsCorrectionshttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:15591839pubmed:commentsCorrectionshttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:15591839pubmed:commentsCorrectionshttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:15591839pubmed:commentsCorrectionshttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:15591839pubmed:commentsCorrectionshttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:15591839pubmed:commentsCorrectionshttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:15591839pubmed:languageenglld:pubmed
pubmed-article:15591839pubmed:journalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:15591839pubmed:citationSubsetIMlld:pubmed
pubmed-article:15591839pubmed:statusMEDLINElld:pubmed
pubmed-article:15591839pubmed:monthDeclld:pubmed
pubmed-article:15591839pubmed:issn0023-4001lld:pubmed
pubmed-article:15591839pubmed:authorpubmed-author:YongTai-SoonT...lld:pubmed
pubmed-article:15591839pubmed:authorpubmed-author:ShinMyeong...lld:pubmed
pubmed-article:15591839pubmed:authorpubmed-author:ParkSoon-Jung...lld:pubmed
pubmed-article:15591839pubmed:authorpubmed-author:KimKyeong...lld:pubmed
pubmed-article:15591839pubmed:authorpubmed-author:SimSeoboSlld:pubmed
pubmed-article:15591839pubmed:authorpubmed-author:ImKyung-ilKIlld:pubmed
pubmed-article:15591839pubmed:issnTypePrintlld:pubmed
pubmed-article:15591839pubmed:volume42lld:pubmed
pubmed-article:15591839pubmed:ownerNLMlld:pubmed
pubmed-article:15591839pubmed:authorsCompleteYlld:pubmed
pubmed-article:15591839pubmed:pagination205-8lld:pubmed
pubmed-article:15591839pubmed:dateRevised2009-11-18lld:pubmed
pubmed-article:15591839pubmed:meshHeadingpubmed-meshheading:15591839...lld:pubmed
pubmed-article:15591839pubmed:meshHeadingpubmed-meshheading:15591839...lld:pubmed
pubmed-article:15591839pubmed:meshHeadingpubmed-meshheading:15591839...lld:pubmed
pubmed-article:15591839pubmed:meshHeadingpubmed-meshheading:15591839...lld:pubmed
pubmed-article:15591839pubmed:meshHeadingpubmed-meshheading:15591839...lld:pubmed
pubmed-article:15591839pubmed:meshHeadingpubmed-meshheading:15591839...lld:pubmed
pubmed-article:15591839pubmed:year2004lld:pubmed
pubmed-article:15591839pubmed:articleTitleUltrastructural observation of human neutrophils during apoptotic cell death triggered by Entamoeba histolytica.lld:pubmed
pubmed-article:15591839pubmed:affiliationDepartment of Parasitology, Institute of Tropical Medicine, and Brain Korea 21 for Medical Science, Yonsei University College of Medicine, Seoul 120-752, Korea.lld:pubmed
pubmed-article:15591839pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:15591839pubmed:publicationTypeIn Vitrolld:pubmed
pubmed-article:15591839pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed
http://linkedlifedata.com/r...pubmed:referesTopubmed-article:15591839lld:pubmed
http://linkedlifedata.com/r...pubmed:referesTopubmed-article:15591839lld:pubmed
http://linkedlifedata.com/r...pubmed:referesTopubmed-article:15591839lld:pubmed