15569824

Source:http://linkedlifedata.com/resource/pubmed/id/15569824

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0011209, umls-concept:C0020507, umls-concept:C0694634, umls-concept:C0694888
pubmed:issue	2
pubmed:dateCreated	2005-1-31
pubmed:abstractText	Phosphoinositide (PI) 3-kinase promotes vascular smooth muscle cell (VSMC) responses necessary for neointimal hyperplasia. We recently demonstrated that the inositol 3-phosphatase PTEN is expressed in VSMCs and that its overexpression inhibits these cellular responses. The purpose of this study was to determine the effects of adenovirus-mediated overexpression of PTEN on neointimal hyperplasia in vivo in the rat carotid injury model.
pubmed:grant	http://linkedlifedata.com/resource/pubmed/grant/HL03557
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/9505803
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/PTEN Phosphohydrolase, http://linkedlifedata.com/resource/pubmed/chemical/PTEN protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Phosphatidylinositol 3-Kinases, http://linkedlifedata.com/resource/pubmed/chemical/Phosphoric Monoester Hydrolases, http://linkedlifedata.com/resource/pubmed/chemical/Recombinant Fusion Proteins, http://linkedlifedata.com/resource/pubmed/chemical/Tumor Suppressor Proteins
pubmed:status	MEDLINE
pubmed:month	Feb
pubmed:issn	1524-4636
pubmed:author	pubmed-author:AnnexBrian HBH, pubmed-author:HuangJianhuaJ, pubmed-author:KontosChristopher DCD, pubmed-author:NiuXi-LinXL, pubmed-author:PippenAnne MAM
pubmed:issnType	Electronic
pubmed:volume	25
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	354-8
pubmed:dateRevised	2010-11-18
pubmed:meshHeading	pubmed-meshheading:15569824-Adenoviridae, pubmed-meshheading:15569824-Animals, pubmed-meshheading:15569824-Apoptosis, pubmed-meshheading:15569824-Balloon Dilation, pubmed-meshheading:15569824-Carotid Artery Injuries, pubmed-meshheading:15569824-Cell Division, pubmed-meshheading:15569824-Gene Therapy, pubmed-meshheading:15569824-Genetic Vectors, pubmed-meshheading:15569824-Humans, pubmed-meshheading:15569824-Hyperplasia, pubmed-meshheading:15569824-Injections, Intra-Arterial, pubmed-meshheading:15569824-Male, pubmed-meshheading:15569824-PTEN Phosphohydrolase, pubmed-meshheading:15569824-Phosphatidylinositol 3-Kinases, pubmed-meshheading:15569824-Phosphoric Monoester Hydrolases, pubmed-meshheading:15569824-Rats, pubmed-meshheading:15569824-Rats, Sprague-Dawley, pubmed-meshheading:15569824-Recombinant Fusion Proteins, pubmed-meshheading:15569824-Signal Transduction, pubmed-meshheading:15569824-Tumor Suppressor Proteins, pubmed-meshheading:15569824-Tunica Intima
pubmed:year	2005
pubmed:articleTitle	Adenovirus-mediated intraarterial delivery of PTEN inhibits neointimal hyperplasia.
pubmed:affiliation	Department of Medicine, Duke University Medical Center, Durham, NC 27710, USA.
pubmed:publicationType	Journal Article, Research Support, U.S. Gov't, P.H.S., Research Support, Non-U.S. Gov't, Research Support, N.I.H., Extramural