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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
5
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pubmed:dateCreated |
2004-11-2
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pubmed:abstractText |
Lymphotoxin is a tumor necrosis factor-family cytokine. Blocking of lymphotoxin alpha 1 beta 2 /lymphotoxin-beta receptor interactions prevents experimental colitis in mice, and this suggests a potential treatment principle of human inflammatory bowel disease. Infection of mice with Citrobacter rodentium serves as an animal model for human infectious colitis induced by enteropathogenic Escherichia coli . We studied the role of lymphotoxin alpha 1 beta 2 /lymphotoxin-beta receptor signaling in Citrobacter rodentium -induced colitis.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Nov
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pubmed:issn |
0016-5085
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
127
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1463-73
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:15521015-Animals,
pubmed-meshheading:15521015-Citrobacter rodentium,
pubmed-meshheading:15521015-Colitis,
pubmed-meshheading:15521015-Dendritic Cells,
pubmed-meshheading:15521015-Disease Models, Animal,
pubmed-meshheading:15521015-Enterobacteriaceae Infections,
pubmed-meshheading:15521015-Flow Cytometry,
pubmed-meshheading:15521015-Immunity, Mucosal,
pubmed-meshheading:15521015-Immunoglobulin G,
pubmed-meshheading:15521015-Immunohistochemistry,
pubmed-meshheading:15521015-Lymphotoxin beta Receptor,
pubmed-meshheading:15521015-Mice,
pubmed-meshheading:15521015-Mice, Inbred C57BL,
pubmed-meshheading:15521015-Receptors, Tumor Necrosis Factor,
pubmed-meshheading:15521015-Recombinant Fusion Proteins
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pubmed:year |
2004
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pubmed:articleTitle |
The lymphotoxin-beta receptor is critical for control of murine Citrobacter rodentium-induced colitis.
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pubmed:affiliation |
Department of Medicine, Münster University Hospital, Münster, Germany. spahn@uni-muenster.de
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't
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