Linked Life Data

15492323

Source:http://linkedlifedata.com/resource/pubmed/id/15492323

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
17
pubmed:dateCreated
2004-10-26
pubmed:abstractText
Although downregulation of L-type Ca2+ current (I(Ca,L)) in chronic atrial fibrillation (AF) is an important determinant of electrical remodeling, the molecular mechanisms are not fully understood. Here, we tested whether reduced I(Ca,L) in AF is associated with alterations in phosphorylation-dependent channel regulation.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Oct
pubmed:issn
1524-4539
pubmed:author
pubmed:issnType
Electronic
pubmed:day
26
pubmed:volume
110
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
2651-7
pubmed:dateRevised
2009-11-19
pubmed:meshHeading
pubmed:year
2004
pubmed:articleTitle
L-type Ca2+ current downregulation in chronic human atrial fibrillation is associated with increased activity of protein phosphatases.
pubmed:affiliation
Department of Pharmacology,Dresden University of Technology, Dresden, Germany.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't