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rdf:type |
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|
lifeskim:mentions |
|
|
pubmed:issue |
5
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|
pubmed:dateCreated |
1992-4-22
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|
pubmed:abstractText |
The 21 kd NusG protein is essential for E. coli viability. Cells depleted for NusG were defective for factor-dependent transcription termination. Rho-induced polarity in the gal operon and the Rho-dependent lambda tR1 and lambda tL1 terminators were suppressed in NusG-deficient cells. NusG depletion inactivated the phage HK022 Nun termination factor. In contrast, the factor-independent lambda tl terminator was fully active in NusG-depleted cells and could be suppressed by phage lambda N function.
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pubmed:grant |
|
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pubmed:language |
eng
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|
pubmed:journal |
|
|
pubmed:citationSubset |
IM
|
|
pubmed:chemical |
|
|
pubmed:status |
MEDLINE
|
|
pubmed:month |
Mar
|
|
pubmed:issn |
0092-8674
|
|
pubmed:author |
|
|
pubmed:issnType |
Print
|
|
pubmed:day |
6
|
|
pubmed:volume |
68
|
|
pubmed:owner |
NLM
|
|
pubmed:authorsComplete |
Y
|
|
pubmed:pagination |
989-94
|
|
pubmed:dateRevised |
2007-11-14
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|
pubmed:meshHeading |
|
|
pubmed:year |
1992
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|
pubmed:articleTitle |
Requirement for E. coli NusG protein in factor-dependent transcription termination.
|
|
pubmed:affiliation |
Institute of Cancer Research, College of Physicians and Surgeons, Columbia University, New York, New York 10032.
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|
pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.
|
