Source:http://linkedlifedata.com/resource/pubmed/id/15371767
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
4 Pt 2
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pubmed:dateCreated |
2004-9-16
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pubmed:abstractText |
Recent data suggest that aldosterone directly mediates cardiac fibrosis and hypertensive nephrosclerosis. We conducted experiments to determine whether administration of spironolactone, a mineralocorticoid receptor antagonist, reduced renal fibrosis in an experimental model of obstructive uropathy.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
AIM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Oct
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pubmed:issn |
0022-5347
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
172
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1590-4
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pubmed:meshHeading |
pubmed-meshheading:15371767-Aldosterone Antagonists,
pubmed-meshheading:15371767-Animals,
pubmed-meshheading:15371767-Fibrosis,
pubmed-meshheading:15371767-Kidney,
pubmed-meshheading:15371767-Male,
pubmed-meshheading:15371767-Mice,
pubmed-meshheading:15371767-Mice, Inbred C57BL,
pubmed-meshheading:15371767-Spironolactone
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pubmed:year |
2004
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pubmed:articleTitle |
Prevention of renal fibrosis by spironolactone in mice with complete unilateral ureteral obstruction.
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pubmed:affiliation |
Department of Pediatrics, Schneider Children's Hospital, New Hyde Park, New York 11040, USA. trachtma@lij.edu
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pubmed:publicationType |
Journal Article
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