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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
3
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pubmed:dateCreated |
2004-8-16
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pubmed:abstractText |
Prostaglandins (PGs) are suggested to be involved in the pathophysiology of different bladder disorders and it has been demonstrated that cyclooxygenase-2 expression is increased as a consequence of bladder outflow obstruction. We investigated whether the PGE2 receptor EP1 is involved in the regulation of normal micturition, the response to intravesical PGE2 administration, and the development of bladder hypertrophy and overactivity due to bladder outlet obstruction (BOO).
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Sep
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pubmed:issn |
0022-5347
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
172
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1166-70
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pubmed:dateRevised |
2010-11-18
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pubmed:meshHeading |
pubmed-meshheading:15311063-Animals,
pubmed-meshheading:15311063-Dinoprostone,
pubmed-meshheading:15311063-Female,
pubmed-meshheading:15311063-Mice,
pubmed-meshheading:15311063-Mice, Knockout,
pubmed-meshheading:15311063-Muscle Contraction,
pubmed-meshheading:15311063-Organ Size,
pubmed-meshheading:15311063-Receptors, Prostaglandin E,
pubmed-meshheading:15311063-Receptors, Prostaglandin E, EP1 Subtype,
pubmed-meshheading:15311063-Urinary Bladder,
pubmed-meshheading:15311063-Urinary Bladder Neck Obstruction,
pubmed-meshheading:15311063-Urination,
pubmed-meshheading:15311063-Urodynamics
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pubmed:year |
2004
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pubmed:articleTitle |
Detrusor responses to prostaglandin E2 and bladder outlet obstruction in wild-type and Ep1 receptor knockout mice.
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pubmed:affiliation |
Department of Clinical Pharmacology, Lund University Hospital, 221 85 Lund, Sweden.
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pubmed:publicationType |
Journal Article
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