Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
7
pubmed:dateCreated
2004-7-12
pubmed:abstractText
To reveal the cause of the impaired elimination of Salmonella enteritidis in HLA-B27-transfected human monocytic cells and to study whether the B pocket of HLA-B27 contributes to these modulatory effects.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jul
pubmed:issn
0004-3591
pubmed:author
pubmed:issnType
Print
pubmed:volume
50
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
2255-63
pubmed:dateRevised
2007-11-14
pubmed:meshHeading
pubmed-meshheading:15248225-Amino Acid Sequence, pubmed-meshheading:15248225-Amino Acid Substitution, pubmed-meshheading:15248225-Cell Line, pubmed-meshheading:15248225-Cell Membrane, pubmed-meshheading:15248225-Endoplasmic Reticulum, pubmed-meshheading:15248225-Gene Transfer Techniques, pubmed-meshheading:15248225-Green Fluorescent Proteins, pubmed-meshheading:15248225-HLA-B27 Antigen, pubmed-meshheading:15248225-Histocompatibility Antigens Class I, pubmed-meshheading:15248225-Humans, pubmed-meshheading:15248225-Intracellular Membranes, pubmed-meshheading:15248225-Luminescent Proteins, pubmed-meshheading:15248225-Methionine, pubmed-meshheading:15248225-Monocytes, pubmed-meshheading:15248225-Phenotype, pubmed-meshheading:15248225-Protein Folding, pubmed-meshheading:15248225-Salmonella Infections, pubmed-meshheading:15248225-Salmonella enteritidis, pubmed-meshheading:15248225-Time Factors, pubmed-meshheading:15248225-Transfection
pubmed:year
2004
pubmed:articleTitle
Enhanced intracellular replication of Salmonella enteritidis in HLA-B27-expressing human monocytic cells: dependency on glutamic acid at position 45 in the B pocket of HLA-B27.
pubmed:affiliation
Department of Human Microbial Ecology and Inflammation, National Public Health Institute, Turku, Finland. markus.penttinen@utu.fi
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, P.H.S., Research Support, Non-U.S. Gov't