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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
6
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pubmed:dateCreated |
2004-5-31
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pubmed:abstractText |
Myocardial infarction (MI) promotes deleterious remodeling of the myocardium, resulting in ventricular dilation and pump dysfunction. Supplementing infarcted myocardium with neonatal myocyte would attenuate deleterious remodeling and so the present study used Doppler echocardiography and histology to analyze the cardiac function and histological regeneration of the damaged myocardium after cellular cardiomyoplasty.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Jun
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pubmed:issn |
1346-9843
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:volume |
68
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
580-6
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pubmed:dateRevised |
2011-11-17
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pubmed:meshHeading |
pubmed-meshheading:15170096-Actin Cytoskeleton,
pubmed-meshheading:15170096-Animals,
pubmed-meshheading:15170096-Cardiomyoplasty,
pubmed-meshheading:15170096-Cell Transplantation,
pubmed-meshheading:15170096-Connexin 43,
pubmed-meshheading:15170096-Disease Models, Animal,
pubmed-meshheading:15170096-Echocardiography, Doppler,
pubmed-meshheading:15170096-Heart Function Tests,
pubmed-meshheading:15170096-Immunohistochemistry,
pubmed-meshheading:15170096-Microscopy, Electron,
pubmed-meshheading:15170096-Muscle Cells,
pubmed-meshheading:15170096-Myocardial Infarction,
pubmed-meshheading:15170096-Rats,
pubmed-meshheading:15170096-Rats, Inbred Lew,
pubmed-meshheading:15170096-Regeneration,
pubmed-meshheading:15170096-Ventricular Remodeling
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pubmed:year |
2004
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pubmed:articleTitle |
Effects of cellular cardiomyoplasty on ventricular remodeling assessed by Doppler echocardiography and topographic immunohistochemistry.
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pubmed:affiliation |
Department of Internal Medicine and Cardiology, Graduate School of Medicine, Osaka City University, Japan.
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pubmed:publicationType |
Journal Article
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