rdf:type |
|
lifeskim:mentions |
umls-concept:C0001272,
umls-concept:C0012984,
umls-concept:C0178539,
umls-concept:C0205171,
umls-concept:C0205173,
umls-concept:C0205252,
umls-concept:C0234402,
umls-concept:C0441712,
umls-concept:C0443199,
umls-concept:C0596981,
umls-concept:C0680635,
umls-concept:C1522565,
umls-concept:C2926735
|
pubmed:issue |
3
|
pubmed:dateCreated |
1992-10-6
|
pubmed:abstractText |
We tested the hypothesis that action potential duration (APD) restitution of normal ventricular muscle cells is different during double premature stimuli (S3) compared with a single premature stimulus (S2). We propose a possible ionic mechanism for such a difference.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
AIM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Sep
|
pubmed:issn |
0009-7322
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
86
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
955-67
|
pubmed:dateRevised |
2010-3-24
|
pubmed:meshHeading |
pubmed-meshheading:1516208-Action Potentials,
pubmed-meshheading:1516208-Animals,
pubmed-meshheading:1516208-Caffeine,
pubmed-meshheading:1516208-Calcium,
pubmed-meshheading:1516208-Dogs,
pubmed-meshheading:1516208-Female,
pubmed-meshheading:1516208-Male,
pubmed-meshheading:1516208-Myocardial Contraction,
pubmed-meshheading:1516208-Papillary Muscles,
pubmed-meshheading:1516208-Reaction Time,
pubmed-meshheading:1516208-Refractory Period, Electrophysiological,
pubmed-meshheading:1516208-Ryanodine,
pubmed-meshheading:1516208-Stimulation, Chemical
|
pubmed:year |
1992
|
pubmed:articleTitle |
Cellular mechanisms of differential action potential duration restitution in canine ventricular muscle cells during single versus double premature stimuli.
|
pubmed:affiliation |
Cedars-Sinai Research Institute, Cedars-Sinai Medical Center, Los Angeles, CA 90048.
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|