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pubmed-article:15146427 | lifeskim:mentions | umls-concept:C0332307 | lld:lifeskim |
pubmed-article:15146427 | lifeskim:mentions | umls-concept:C0009325 | lld:lifeskim |
pubmed-article:15146427 | lifeskim:mentions | umls-concept:C0011644 | lld:lifeskim |
pubmed-article:15146427 | lifeskim:mentions | umls-concept:C0597295 | lld:lifeskim |
pubmed-article:15146427 | lifeskim:mentions | umls-concept:C0076930 | lld:lifeskim |
pubmed-article:15146427 | lifeskim:mentions | umls-concept:C0021467 | lld:lifeskim |
pubmed-article:15146427 | lifeskim:mentions | umls-concept:C0456603 | lld:lifeskim |
pubmed-article:15146427 | lifeskim:mentions | umls-concept:C0205217 | lld:lifeskim |
pubmed-article:15146427 | lifeskim:mentions | umls-concept:C0332325 | lld:lifeskim |
pubmed-article:15146427 | lifeskim:mentions | umls-concept:C0021469 | lld:lifeskim |
pubmed-article:15146427 | lifeskim:mentions | umls-concept:C0205250 | lld:lifeskim |
pubmed-article:15146427 | lifeskim:mentions | umls-concept:C1880177 | lld:lifeskim |
pubmed-article:15146427 | pubmed:issue | 5 | lld:pubmed |
pubmed-article:15146427 | pubmed:dateCreated | 2004-5-17 | lld:pubmed |
pubmed-article:15146427 | pubmed:abstractText | Aberrant transforming growth factor beta (TGFbeta) signaling has been implicated in the pathogenesis of scleroderma (systemic sclerosis [SSc]), but the contribution of specific components in this pathway to SSc fibroblast phenotype remains unclear. This study was undertaken to delineate the role of TGFbeta receptor type I (TGFbetaRI) and TGFbetaRII in collagen overexpression by SSc fibroblasts. | lld:pubmed |
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pubmed-article:15146427 | pubmed:language | eng | lld:pubmed |
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pubmed-article:15146427 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:15146427 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:15146427 | pubmed:month | May | lld:pubmed |
pubmed-article:15146427 | pubmed:issn | 0004-3591 | lld:pubmed |
pubmed-article:15146427 | pubmed:author | pubmed-author:SmithEdwin... | lld:pubmed |
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pubmed-article:15146427 | pubmed:author | pubmed-author:BlaszczykMari... | lld:pubmed |
pubmed-article:15146427 | pubmed:author | pubmed-author:PannuJaspreet... | lld:pubmed |
pubmed-article:15146427 | pubmed:author | pubmed-author:YamanakaMasay... | lld:pubmed |
pubmed-article:15146427 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:15146427 | pubmed:volume | 50 | lld:pubmed |
pubmed-article:15146427 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:15146427 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:15146427 | pubmed:pagination | 1566-77 | lld:pubmed |
pubmed-article:15146427 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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pubmed-article:15146427 | pubmed:year | 2004 | lld:pubmed |
pubmed-article:15146427 | pubmed:articleTitle | An increased transforming growth factor beta receptor type I:type II ratio contributes to elevated collagen protein synthesis that is resistant to inhibition via a kinase-deficient transforming growth factor beta receptor type II in scleroderma. | lld:pubmed |
pubmed-article:15146427 | pubmed:affiliation | Division of Rheumatology and Immunology, Medical University of South Carolina, 96 Jonathan Lucas Street, Charleston, SC 29425, USA. | lld:pubmed |
pubmed-article:15146427 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:15146427 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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