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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1
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pubmed:dateCreated |
2004-4-29
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pubmed:abstractText |
The progressive shortening of the atrial effective refractory period (ERP) during atrial fibrillation (AF) might be due to the activation of the KATP channels by rapid atrial rates. We tested the hypothesis that the cardioselective, sarcolemmal KATP channel blocker HMR 1098 would prevent atrial ERP shortening during AF.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Jan
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pubmed:issn |
0920-3206
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
18
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
23-30
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:15115900-Adenosine Triphosphate,
pubmed-meshheading:15115900-Animals,
pubmed-meshheading:15115900-Atrial Fibrillation,
pubmed-meshheading:15115900-Atrial Function, Right,
pubmed-meshheading:15115900-Benzamides,
pubmed-meshheading:15115900-Dogs,
pubmed-meshheading:15115900-Female,
pubmed-meshheading:15115900-Male,
pubmed-meshheading:15115900-Potassium Channel Blockers,
pubmed-meshheading:15115900-Refractory Period, Electrophysiological,
pubmed-meshheading:15115900-Sarcolemma
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pubmed:year |
2004
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pubmed:articleTitle |
Effect of the cardioselective, sarcolemmal K(ATP) channel blocker HMR 1098 on atrial electrical remodeling during pacing-induced atrial fibrillation in dogs.
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pubmed:affiliation |
3rd Department of Medicine, Semmelweis University, School of Medicine, Budapest, Hungary. vereckei@kut.sote.hu
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pubmed:publicationType |
Journal Article
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