Source:http://linkedlifedata.com/resource/pubmed/id/15103251
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
3
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pubmed:dateCreated |
2004-4-22
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pubmed:abstractText |
Successful analysis of the pathophysiology of rheumatoid arthritis requires the functional understanding of interactions between different cell types and the cell matrix, intracellular signaling pathways, as well as between cartilage, bone, and synovium in rheumatoid arthritis. During the review period, molecular biology has provided and used a growing number of tools to screen the genome such as gene and protein chips, haplotype analysis, and single nucleotide polymorphism analysis, resulting in various novel findings with considerable impact on the overall understanding of rheumatoid arthritis.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
May
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pubmed:issn |
1040-8711
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
16
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
238-45
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pubmed:dateRevised |
2005-11-16
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pubmed:meshHeading |
pubmed-meshheading:15103251-Apoptosis,
pubmed-meshheading:15103251-Arthritis, Rheumatoid,
pubmed-meshheading:15103251-Cell Adhesion Molecules,
pubmed-meshheading:15103251-Cell Division,
pubmed-meshheading:15103251-Fibroblasts,
pubmed-meshheading:15103251-Genomics,
pubmed-meshheading:15103251-Humans,
pubmed-meshheading:15103251-Inflammation,
pubmed-meshheading:15103251-Matrix Metalloproteinases,
pubmed-meshheading:15103251-Neovascularization, Pathologic,
pubmed-meshheading:15103251-Signal Transduction
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pubmed:year |
2004
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pubmed:articleTitle |
Functional genomics of fibroblasts.
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pubmed:affiliation |
Department of Internal Medicine I, University of Regensburg, D-93042 Regensburg, Germany. ulf.mueller-ladner@klinik.uni-regensburg.de
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pubmed:publicationType |
Journal Article,
Review
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