| pubmed-article:15086471 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:15086471 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:15086471 | lifeskim:mentions | umls-concept:C0079281 | lld:lifeskim |
| pubmed-article:15086471 | lifeskim:mentions | umls-concept:C0022677 | lld:lifeskim |
| pubmed-article:15086471 | lifeskim:mentions | umls-concept:C0596138 | lld:lifeskim |
| pubmed-article:15086471 | lifeskim:mentions | umls-concept:C1420234 | lld:lifeskim |
| pubmed-article:15086471 | lifeskim:mentions | umls-concept:C0851285 | lld:lifeskim |
| pubmed-article:15086471 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:15086471 | pubmed:dateCreated | 2004-4-16 | lld:pubmed |
| pubmed-article:15086471 | pubmed:abstractText | Chronic metabolic acidosis leads to an increase in NHE3 activity that is mediated by endothelin-1 (ET-1) expression and activation of the proximal tubule endothelin B receptor. Chronic metabolic acidosis increases preproET-1 mRNA abundance in kidney cortex, but the cell responsible has not been identified. | lld:pubmed |
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| pubmed-article:15086471 | pubmed:language | eng | lld:pubmed |
| pubmed-article:15086471 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15086471 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:15086471 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15086471 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:15086471 | pubmed:issn | 0085-2538 | lld:pubmed |
| pubmed-article:15086471 | pubmed:author | pubmed-author:AlpernRobert... | lld:pubmed |
| pubmed-article:15086471 | pubmed:author | pubmed-author:YanagisawaMas... | lld:pubmed |
| pubmed-article:15086471 | pubmed:author | pubmed-author:LichtChristop... | lld:pubmed |
| pubmed-article:15086471 | pubmed:author | pubmed-author:LaghmaniKamel... | lld:pubmed |
| pubmed-article:15086471 | pubmed:author | pubmed-author:PreisigPatric... | lld:pubmed |
| pubmed-article:15086471 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:15086471 | pubmed:volume | 65 | lld:pubmed |
| pubmed-article:15086471 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15086471 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15086471 | pubmed:pagination | 1320-6 | lld:pubmed |
| pubmed-article:15086471 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:15086471 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:15086471 | pubmed:articleTitle | An autocrine role for endothelin-1 in the regulation of proximal tubule NHE3. | lld:pubmed |
| pubmed-article:15086471 | pubmed:affiliation | Department of Internal Medicine and Howard Hughes Medical Institute, University of Texas Southwestern Medical Center, Dallas, Texas 75390, USA. | lld:pubmed |
| pubmed-article:15086471 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15086471 | pubmed:publicationType | In Vitro | lld:pubmed |
| pubmed-article:15086471 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:15086471 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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