Source:http://linkedlifedata.com/resource/pubmed/id/15084771
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
3
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pubmed:dateCreated |
2004-4-15
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pubmed:abstractText |
Physicochemical properties of urine do not explain the formation of urinary stones. Clinical findings and results of animal experiments suggest that alteration to the renal tubular cell plays a key role in the initiation of urinary stone formation. It is not clear whether this is a primarily intracellular alteration of metabolic origin which, after lysis of the renal tubular cell in the lumen, presents a nucleus for the formation of concretions, or whether in the lumen it is tubular cell damage induced by crystalluria that triggers the formation of urinary stones.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:status |
MEDLINE
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pubmed:issn |
0042-1138
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pubmed:author | |
pubmed:copyrightInfo |
Copyright 2004 S. Karger AG, Basel
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pubmed:issnType |
Print
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pubmed:volume |
72
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
244-51
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pubmed:dateRevised |
2006-10-30
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pubmed:meshHeading | |
pubmed:year |
2004
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pubmed:articleTitle |
Renal tubular alteration by crystalluria in stone disease-an experimental study by means of MDCK cells.
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pubmed:affiliation |
Department of Urology, University of Tübingen, Tübingen, Germany. sven.lahme@med.uni-tuebingen.de
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pubmed:publicationType |
Journal Article
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