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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
4
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pubmed:dateCreated |
2004-4-8
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pubmed:abstractText |
Previous studies undertaken by the authors have indicated that iron accumulation and oxidative stress in the brain contribute to secondary brain damage after intracerebral hemorrhage (ICH). In the present study the authors investigate whether deferoxamine, an iron chelator, can reduce ICH-induced brain injury.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Apr
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pubmed:issn |
0022-3085
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
100
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
672-8
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:15070122-Animals,
pubmed-meshheading:15070122-Blotting, Western,
pubmed-meshheading:15070122-Brain Edema,
pubmed-meshheading:15070122-Cerebral Hemorrhage,
pubmed-meshheading:15070122-DNA Damage,
pubmed-meshheading:15070122-Deferoxamine,
pubmed-meshheading:15070122-Disease Models, Animal,
pubmed-meshheading:15070122-Hematoma,
pubmed-meshheading:15070122-Humans,
pubmed-meshheading:15070122-Iron,
pubmed-meshheading:15070122-Iron Chelating Agents,
pubmed-meshheading:15070122-Male,
pubmed-meshheading:15070122-Oxidative Stress,
pubmed-meshheading:15070122-Rats,
pubmed-meshheading:15070122-Rats, Sprague-Dawley
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pubmed:year |
2004
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pubmed:articleTitle |
Deferoxamine-induced attenuation of brain edema and neurological deficits in a rat model of intracerebral hemorrhage.
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pubmed:affiliation |
Department of Neurosurgery, University of Michigan, Ann Arbor, Michigan 48109-0532, USA.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.
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