Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
14
pubmed:dateCreated
2004-4-13
pubmed:abstractText
Circulating endothelial progenitor cells (EPCs) have been reported previously. In this study, we examined the hypothesis that overexpression of vasculoprotective gene endothelial nitric oxide synthase (eNOS) and heme oxygenase-1 (HO-1) in EPCs enhances their ability to inhibit neointimal hyperplasia.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
http://linkedlifedata.com/resource/pubmed/chemical/DNA, Complementary, http://linkedlifedata.com/resource/pubmed/chemical/Green Fluorescent Proteins, http://linkedlifedata.com/resource/pubmed/chemical/HMOX1 protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Heme Oxygenase (Decyclizing), http://linkedlifedata.com/resource/pubmed/chemical/Heme Oxygenase-1, http://linkedlifedata.com/resource/pubmed/chemical/Luminescent Proteins, http://linkedlifedata.com/resource/pubmed/chemical/Membrane Proteins, http://linkedlifedata.com/resource/pubmed/chemical/NOS3 protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Nitric Oxide Synthase, http://linkedlifedata.com/resource/pubmed/chemical/Nitric Oxide Synthase Type III, http://linkedlifedata.com/resource/pubmed/chemical/RNA, Messenger, http://linkedlifedata.com/resource/pubmed/chemical/Recombinant Fusion Proteins
pubmed:status
MEDLINE
pubmed:month
Apr
pubmed:issn
1524-4539
pubmed:author
pubmed:issnType
Electronic
pubmed:day
13
pubmed:volume
109
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
1769-75
pubmed:dateRevised
2007-11-14
pubmed:meshHeading
pubmed-meshheading:15066951-Angioplasty, Balloon, pubmed-meshheading:15066951-Animals, pubmed-meshheading:15066951-Carotid Artery, Common, pubmed-meshheading:15066951-Carotid Artery Injuries, pubmed-meshheading:15066951-Cells, Cultured, pubmed-meshheading:15066951-DNA, Complementary, pubmed-meshheading:15066951-Endothelial Cells, pubmed-meshheading:15066951-Endothelium, Vascular, pubmed-meshheading:15066951-Enzyme Induction, pubmed-meshheading:15066951-Gene Therapy, pubmed-meshheading:15066951-Genes, Reporter, pubmed-meshheading:15066951-Genetic Vectors, pubmed-meshheading:15066951-Green Fluorescent Proteins, pubmed-meshheading:15066951-Heme Oxygenase (Decyclizing), pubmed-meshheading:15066951-Heme Oxygenase-1, pubmed-meshheading:15066951-Humans, pubmed-meshheading:15066951-Hyperplasia, pubmed-meshheading:15066951-Luminescent Proteins, pubmed-meshheading:15066951-Male, pubmed-meshheading:15066951-Membrane Proteins, pubmed-meshheading:15066951-Nitric Oxide Synthase, pubmed-meshheading:15066951-Nitric Oxide Synthase Type III, pubmed-meshheading:15066951-RNA, Messenger, pubmed-meshheading:15066951-Rabbits, pubmed-meshheading:15066951-Recombinant Fusion Proteins, pubmed-meshheading:15066951-Retroviridae, pubmed-meshheading:15066951-Thrombosis, pubmed-meshheading:15066951-Transduction, Genetic, pubmed-meshheading:15066951-Tunica Intima
pubmed:year
2004
pubmed:articleTitle
Enhanced inhibition of neointimal hyperplasia by genetically engineered endothelial progenitor cells.
pubmed:affiliation
Department of Medicine, Brigham and Women's Hospital, Harvard Medical School, Boston, Mass 02115, USA.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, P.H.S., Research Support, Non-U.S. Gov't, Evaluation Studies