|
rdf:type |
|
|
lifeskim:mentions |
|
|
pubmed:issue |
5
|
|
pubmed:dateCreated |
2004-3-15
|
|
pubmed:abstractText |
Warm ischemia-reperfusion (I/R) injury plays an important role in posttransplant organ failure. In particular, organs from marginal donors suffer I/R injury. Although iron has been implicated in the pathophysiology of renal I/R injury, the mechanism of iron-mediated injury remains to be established. The authors therefore investigated the role of circulating redox-active iron in an experimental model for renal I/R injury.
|
|
pubmed:language |
eng
|
|
pubmed:journal |
|
|
pubmed:citationSubset |
IM
|
|
pubmed:chemical |
|
|
pubmed:status |
MEDLINE
|
|
pubmed:month |
Mar
|
|
pubmed:issn |
0041-1337
|
|
pubmed:author |
|
|
pubmed:issnType |
Print
|
|
pubmed:day |
15
|
|
pubmed:volume |
77
|
|
pubmed:owner |
NLM
|
|
pubmed:authorsComplete |
Y
|
|
pubmed:pagination |
669-75
|
|
pubmed:dateRevised |
2011-11-17
|
|
pubmed:meshHeading |
pubmed-meshheading:15021827-Animals,
pubmed-meshheading:15021827-Apoproteins,
pubmed-meshheading:15021827-Apoptosis,
pubmed-meshheading:15021827-Complement Activation,
pubmed-meshheading:15021827-Iron,
pubmed-meshheading:15021827-Kidney,
pubmed-meshheading:15021827-Male,
pubmed-meshheading:15021827-Mice,
pubmed-meshheading:15021827-Nephrectomy,
pubmed-meshheading:15021827-Neutrophils,
pubmed-meshheading:15021827-Oxidation-Reduction,
pubmed-meshheading:15021827-Reactive Oxygen Species,
pubmed-meshheading:15021827-Reperfusion Injury,
pubmed-meshheading:15021827-Transferrin,
pubmed-meshheading:15021827-Transplants
|
|
pubmed:year |
2004
|
|
pubmed:articleTitle |
Reduction of circulating redox-active iron by apotransferrin protects against renal ischemia-reperfusion injury.
|
|
pubmed:affiliation |
Department of Surgery, Nutrition and Toxicology Research Institute Maastricht, Academic Hospital Maastricht, Maastricht, The Netherlands.
|
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|
