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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
3 Pt 1
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pubmed:dateCreated |
2004-3-2
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pubmed:abstractText |
Many extremely preterm infants develop hyperglycemia in the first week of life during continuous glucose infusion. The objective of this study was to determine whether defective insulin secretion or resistance to insulin was the primary factor involved in transient hyperglycemia of extremely preterm infants.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Mar
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pubmed:issn |
1098-4275
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:volume |
113
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
537-41
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pubmed:dateRevised |
2011-11-17
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pubmed:meshHeading |
pubmed-meshheading:14993546-Blood Glucose,
pubmed-meshheading:14993546-C-Peptide,
pubmed-meshheading:14993546-Female,
pubmed-meshheading:14993546-Humans,
pubmed-meshheading:14993546-Hyperglycemia,
pubmed-meshheading:14993546-Infant, Newborn,
pubmed-meshheading:14993546-Infant, Premature,
pubmed-meshheading:14993546-Infant, Premature, Diseases,
pubmed-meshheading:14993546-Insulin,
pubmed-meshheading:14993546-Insulin Resistance,
pubmed-meshheading:14993546-Islets of Langerhans,
pubmed-meshheading:14993546-Male,
pubmed-meshheading:14993546-Proinsulin,
pubmed-meshheading:14993546-Prospective Studies
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pubmed:year |
2004
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pubmed:articleTitle |
Both relative insulin resistance and defective islet beta-cell processing of proinsulin are responsible for transient hyperglycemia in extremely preterm infants.
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pubmed:affiliation |
Service de Réanimation Pédiatrique et Néonatale, Hôpital Necker-Enfants Malades, Paris, France. delphine.mitanchez@nck.ap-hop-paris.fr
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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