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| pubmed-article:14966779 | lifeskim:mentions | umls-concept:C0018801 | lld:lifeskim |
| pubmed-article:14966779 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:14966779 | lifeskim:mentions | umls-concept:C0021467 | lld:lifeskim |
| pubmed-article:14966779 | lifeskim:mentions | umls-concept:C1366499 | lld:lifeskim |
| pubmed-article:14966779 | lifeskim:mentions | umls-concept:C0021469 | lld:lifeskim |
| pubmed-article:14966779 | lifeskim:mentions | umls-concept:C1704243 | lld:lifeskim |
| pubmed-article:14966779 | lifeskim:mentions | umls-concept:C1554184 | lld:lifeskim |
| pubmed-article:14966779 | lifeskim:mentions | umls-concept:C1999230 | lld:lifeskim |
| pubmed-article:14966779 | lifeskim:mentions | umls-concept:C1958507 | lld:lifeskim |
| pubmed-article:14966779 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:14966779 | pubmed:dateCreated | 2004-2-17 | lld:pubmed |
| pubmed-article:14966779 | pubmed:abstractText | Vasopeptidase inhibitors (VPi) may provide a new means of treating hypertension and congestive heart failure, because they simultaneously block angiotensin-converting enzyme (ACE) and neutral endopeptidase-24.11 (NEP-24.11), thereby inhibiting the renin-angiotensin system and enhancing vasodilator and natriuretic substances such as kinins and natriuretic peptides. | lld:pubmed |
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| pubmed-article:14966779 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:14966779 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:14966779 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:14966779 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:14966779 | pubmed:issn | 1071-9164 | lld:pubmed |
| pubmed-article:14966779 | pubmed:author | pubmed-author:SheselyEdward... | lld:pubmed |
| pubmed-article:14966779 | pubmed:author | pubmed-author:YangXiao-Ping... | lld:pubmed |
| pubmed-article:14966779 | pubmed:author | pubmed-author:CarreteroOsca... | lld:pubmed |
| pubmed-article:14966779 | pubmed:author | pubmed-author:XuJiangJ | lld:pubmed |
| pubmed-article:14966779 | pubmed:author | pubmed-author:LiuYun-HeYH | lld:pubmed |
| pubmed-article:14966779 | pubmed:author | pubmed-author:YangFangF | lld:pubmed |
| pubmed-article:14966779 | pubmed:author | pubmed-author:Oja-TebbeNanc... | lld:pubmed |
| pubmed-article:14966779 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:14966779 | pubmed:volume | 10 | lld:pubmed |
| pubmed-article:14966779 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:14966779 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:14966779 | pubmed:pagination | 83-9 | lld:pubmed |
| pubmed-article:14966779 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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| pubmed-article:14966779 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:14966779 | pubmed:articleTitle | Dual inhibition of ACE and NEP provides greater cardioprotection in mice with heart failure. | lld:pubmed |
| pubmed-article:14966779 | pubmed:affiliation | Hypertension and Vascular Research Division, Henry Ford Hospital, 2799 West Grand Boulevard, Detroit, MI 48202-2689, USA. | lld:pubmed |
| pubmed-article:14966779 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:14966779 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:14966779 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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